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Items: 1 to 20 of 269

1.

Search for the target genes involved in the suppression of antibody production by TCDD in C57BL/6 mice.

Nagai H, Takei T, Tohyama C, Kubo M, Abe R, Nohara K.

Int Immunopharmacol. 2005 Feb;5(2):331-43.

PMID:
15652763
2.

Early consequences of 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure on the activation and survival of antigen-specific T cells.

Funatake CJ, Dearstyne EA, Steppan LB, Shepherd DM, Spanjaard ES, Marshak-Rothstein A, Kerkvliet NI.

Toxicol Sci. 2004 Nov;82(1):129-42. Epub 2004 Aug 13.

3.

Constitutively active aryl hydrocarbon receptor expressed in T cells increases immunization-induced IFN-gamma production in mice but does not suppress T(h)2-cytokine production or antibody production.

Nohara K, Suzuki T, Ao K, Murai H, Miyamoto Y, Inouye K, Pan X, Motohashi H, Fujii-Kuriyama Y, Yamamoto M, Tohyama C.

Int Immunol. 2009 Jul;21(7):769-77. doi: 10.1093/intimm/dxp045. Epub 2009 May 21.

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Analysis of 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced gene expression profile in vivo using pathway-specific cDNA arrays.

Zeytun A, McKallip RJ, Fisher M, Camacho I, Nagarkatti M, Nagarkatti PS.

Toxicology. 2002 Sep 16;178(3):241-60.

PMID:
12167310
9.

Expression of constitutively-active aryl hydrocarbon receptor in T-cells enhances the down-regulation of CD62L, but does not alter expression of CD25 or suppress the allogeneic CTL response.

Funatake CJ, Ao K, Suzuki T, Murai H, Yamamoto M, Fujii-Kuriyama Y, Kerkvliet NI, Nohara K.

J Immunotoxicol. 2009 Sep;6(3):194-203. doi: 10.1080/15476910903124454.

PMID:
19635034
10.

Suppressive effects of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on the high-affinity antibody response in C57BL/6 mice.

Inouye K, Ito T, Fujimaki H, Takahashi Y, Takemori T, Pan X, Tohyama C, Nohara K.

Toxicol Sci. 2003 Aug;74(2):315-24. Epub 2003 May 28.

11.

Low-dose dioxins alter gene expression related to cholesterol biosynthesis, lipogenesis, and glucose metabolism through the aryl hydrocarbon receptor-mediated pathway in mouse liver.

Sato S, Shirakawa H, Tomita S, Ohsaki Y, Haketa K, Tooi O, Santo N, Tohkin M, Furukawa Y, Gonzalez FJ, Komai M.

Toxicol Appl Pharmacol. 2008 May 15;229(1):10-9. doi: 10.1016/j.taap.2007.12.029. Epub 2008 Jan 17.

PMID:
18295293
12.

Aryl hydrocarbon receptor-dependent inhibition of AP-1 activity by 2,3,7,8-tetrachlorodibenzo-p-dioxin in activated B cells.

Suh J, Jeon YJ, Kim HM, Kang JS, Kaminski NE, Yang KH.

Toxicol Appl Pharmacol. 2002 Jun 1;181(2):116-23.

PMID:
12051995
13.

2,3,7,8-Tetrachlorodibenzo-p-dioxin modulates the expression of cKrox and Runx3, transcription regulatory factors controlling the lineage commitment of CD4+CD8+ into CD4 and CD8 thymocytes, respectively.

Gill BC, Jeon CH, Sung HN, Kim HL, Jin DW, Park JH.

Toxicol Lett. 2008 Aug 28;180(3):189-95. doi: 10.1016/j.toxlet.2008.06.856. Epub 2008 Jun 17.

PMID:
18602973
15.

Gene expression profiling in Caco-2 human colon cells exposed to TCDD, benzo[a]pyrene, and natural Ah receptor agonists from cruciferous vegetables and citrus fruits.

de Waard WJ, Aarts JM, Peijnenburg AA, Baykus H, Talsma E, Punt A, de Kok TM, van Schooten FJ, Hoogenboom LA.

Toxicol In Vitro. 2008 Mar;22(2):396-410. Epub 2007 Oct 26.

PMID:
18061397
16.

T cell-derived IL-5 production is a sensitive target of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD).

Inouye K, Pan X, Imai N, Ito T, Takei T, Tohyama C, Nohara K.

Chemosphere. 2005 Aug;60(7):907-13.

PMID:
15992597
17.

Comparison of the 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD)-induced CYP1A1 gene expression profile in lymphocytes from mice, rats, and humans: most potent induction in humans.

Nohara K, Ao K, Miyamoto Y, Ito T, Suzuki T, Toyoshiba H, Tohyama C.

Toxicology. 2006 Aug 15;225(2-3):204-13. Epub 2006 Jun 10.

PMID:
16839655
19.
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