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The ETS family transcription factor PU.1 is necessary for the maintenance of fetal liver hematopoietic stem cells.

Kim HG, de Guzman CG, Swindle CS, Cotta CV, Gartland L, Scott EW, Klug CA.

Blood. 2004 Dec 15;104(13):3894-900. Epub 2004 Aug 24.


Myeloid development is selectively disrupted in PU.1 null mice.

Anderson KL, Smith KA, Conners K, McKercher SR, Maki RA, Torbett BE.

Blood. 1998 May 15;91(10):3702-10.


PU.1 immortalizes hematopoietic progenitors in a GM-CSF-dependent manner.

Houston IB, Huang KJ, Jennings SR, DeKoter RP.

Exp Hematol. 2007 Mar;35(3):374-384.


Commitment to the monocytic lineage occurs in the absence of the transcription factor PU.1.

Henkel GW, McKercher SR, Leenen PJ, Maki RA.

Blood. 1999 May 1;93(9):2849-58.


Spi-C has opposing effects to PU.1 on gene expression in progenitor B cells.

Schweitzer BL, Huang KJ, Kamath MB, Emelyanov AV, Birshtein BK, DeKoter RP.

J Immunol. 2006 Aug 15;177(4):2195-207.


The Hox cofactor and proto-oncogene Pbx1 is required for maintenance of definitive hematopoiesis in the fetal liver.

DiMartino JF, Selleri L, Traver D, Firpo MT, Rhee J, Warnke R, O'Gorman S, Weissman IL, Cleary ML.

Blood. 2001 Aug 1;98(3):618-26.


PU.1 functions in a cell-autonomous manner to control the differentiation of multipotential lymphoid-myeloid progenitors.

Scott EW, Fisher RC, Olson MC, Kehrli EW, Simon MC, Singh H.

Immunity. 1997 Apr;6(4):437-47.


Dynamic regulation of PU.1 expression in multipotent hematopoietic progenitors.

Nutt SL, Metcalf D, D'Amico A, Polli M, Wu L.

J Exp Med. 2005 Jan 17;201(2):221-31.


PU.1 and the granulocyte- and macrophage colony-stimulating factor receptors play distinct roles in late-stage myeloid cell differentiation.

Anderson KL, Smith KA, Perkin H, Hermanson G, Anderson CG, Jolly DJ, Maki RA, Torbett BE.

Blood. 1999 Oct 1;94(7):2310-8.


Transcription factor PU.1 is necessary for development of thymic and myeloid progenitor-derived dendritic cells.

Anderson KL, Perkin H, Surh CD, Venturini S, Maki RA, Torbett BE.

J Immunol. 2000 Feb 15;164(4):1855-61.


Transient expression of PU.1 commits multipotent progenitors to a myeloid fate whereas continued expression favors macrophage over granulocyte differentiation.

McIvor Z, Hein S, Fiegler H, Schroeder T, Stocking C, Just U, Cross M.

Exp Hematol. 2003 Jan;31(1):39-47.


T cell development in PU.1-deficient mice.

Spain LM, Guerriero A, Kunjibettu S, Scott EW.

J Immunol. 1999 Sep 1;163(5):2681-7.


PU.1 but not ets-2 is essential for macrophage development from embryonic stem cells.

Henkel GW, McKercher SR, Yamamoto H, Anderson KL, Oshima RG, Maki RA.

Blood. 1996 Oct 15;88(8):2917-26.


Distinctive and indispensable roles of PU.1 in maintenance of hematopoietic stem cells and their differentiation.

Iwasaki H, Somoza C, Shigematsu H, Duprez EA, Iwasaki-Arai J, Mizuno S, Arinobu Y, Geary K, Zhang P, Dayaram T, Fenyus ML, Elf S, Chan S, Kastner P, Huettner CS, Murray R, Tenen DG, Akashi K.

Blood. 2005 Sep 1;106(5):1590-600. Epub 2005 May 24.


Regulation of macrophage and neutrophil cell fates by the PU.1:C/EBPalpha ratio and granulocyte colony-stimulating factor.

Dahl R, Walsh JC, Lancki D, Laslo P, Iyer SR, Singh H, Simon MC.

Nat Immunol. 2003 Oct;4(10):1029-36. Epub 2003 Sep 7.


Reciprocal activation of GATA-1 and PU.1 marks initial specification of hematopoietic stem cells into myeloerythroid and myelolymphoid lineages.

Arinobu Y, Mizuno S, Chong Y, Shigematsu H, Iino T, Iwasaki H, Graf T, Mayfield R, Chan S, Kastner P, Akashi K.

Cell Stem Cell. 2007 Oct 11;1(4):416-27. doi: 10.1016/j.stem.2007.07.004.


SCL expression at critical points in human hematopoietic lineage commitment.

Zhang Y, Payne KJ, Zhu Y, Price MA, Parrish YK, Zielinska E, Barsky LW, Crooks GM.

Stem Cells. 2005 Jun-Jul;23(6):852-60.

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