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Similar articles for PubMed (Select 15004139)

1.

Murine CD93 (C1qRp) contributes to the removal of apoptotic cells in vivo but is not required for C1q-mediated enhancement of phagocytosis.

Norsworthy PJ, Fossati-Jimack L, Cortes-Hernandez J, Taylor PR, Bygrave AE, Thompson RD, Nourshargh S, Walport MJ, Botto M.

J Immunol. 2004 Mar 15;172(6):3406-14.

2.

Role of surfactant proteins A, D, and C1q in the clearance of apoptotic cells in vivo and in vitro: calreticulin and CD91 as a common collectin receptor complex.

Vandivier RW, Ogden CA, Fadok VA, Hoffmann PR, Brown KK, Botto M, Walport MJ, Fisher JH, Henson PM, Greene KE.

J Immunol. 2002 Oct 1;169(7):3978-86.

3.

Rac2-deficient murine macrophages have selective defects in superoxide production and phagocytosis of opsonized particles.

Yamauchi A, Kim C, Li S, Marchal CC, Towe J, Atkinson SJ, Dinauer MC.

J Immunol. 2004 Nov 15;173(10):5971-9.

4.

Human C1qRp is identical with CD93 and the mNI-11 antigen but does not bind C1q.

McGreal EP, Ikewaki N, Akatsu H, Morgan BP, Gasque P.

J Immunol. 2002 May 15;168(10):5222-32.

5.

Identification of human CD93 as the phagocytic C1q receptor (C1qRp) by expression cloning.

Steinberger P, Szekeres A, Wille S, Stöckl J, Selenko N, Prager E, Staffler G, Madic O, Stockinger H, Knapp W.

J Leukoc Biol. 2002 Jan;71(1):133-40.

6.

IGM is required for efficient complement mediated phagocytosis of apoptotic cells in vivo.

Ogden CA, Kowalewski R, Peng Y, Montenegro V, Elkon KB.

Autoimmunity. 2005 Jun;38(4):259-64.

PMID:
16206508
7.

Lupus-prone mice have an abnormal response to thioglycolate and an impaired clearance of apoptotic cells.

Potter PK, Cortes-Hernandez J, Quartier P, Botto M, Walport MJ.

J Immunol. 2003 Mar 15;170(6):3223-32.

8.

Negative regulation of phagocytosis in macrophages by the CD47-SHPS-1 system.

Okazawa H, Motegi S, Ohyama N, Ohnishi H, Tomizawa T, Kaneko Y, Oldenborg PA, Ishikawa O, Matozaki T.

J Immunol. 2005 Feb 15;174(4):2004-11.

9.

Critical role of galectin-3 in phagocytosis by macrophages.

Sano H, Hsu DK, Apgar JR, Yu L, Sharma BB, Kuwabara I, Izui S, Liu FT.

J Clin Invest. 2003 Aug;112(3):389-97.

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C1qRP is a heavily O-glycosylated cell surface protein involved in the regulation of phagocytic activity.

Nepomuceno RR, Ruiz S, Park M, Tenner AJ.

J Immunol. 1999 Mar 15;162(6):3583-9.

13.

Role of complement component C1q in phagocytosis of Listeria monocytogenes by murine macrophage-like cell lines.

Alvarez-Dominguez C, Carrasco-Marin E, Leyva-Cobian F.

Infect Immun. 1993 Sep;61(9):3664-72.

14.

Detection and characterization of soluble CD93 released during inflammation.

Greenlee MC, Sullivan SA, Bohlson SS.

Inflamm Res. 2009 Dec;58(12):909-19. doi: 10.1007/s00011-009-0064-0. Epub 2009 Jul 15.

PMID:
19603257
15.

Cell surface expression of C1qRP/CD93 is stabilized by O-glycosylation.

Park M, Tenner AJ.

J Cell Physiol. 2003 Sep;196(3):512-22.

PMID:
12891708
16.

Inhibitory effect of Toll-like receptor 4 on fusion between phagosomes and endosomes/lysosomes in macrophages.

Shiratsuchi A, Watanabe I, Takeuchi O, Akira S, Nakanishi Y.

J Immunol. 2004 Feb 15;172(4):2039-47.

17.

Structure-function studies of the receptors for complement C1q.

McGreal E, Gasque P.

Biochem Soc Trans. 2002 Nov;30(Pt 6):1010-4. Review.

PMID:
12440963
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Cutting edge: the Wiskott-Aldrich syndrome protein is required for efficient phagocytosis of apoptotic cells.

Leverrier Y, Lorenzi R, Blundell MP, Brickell P, Kinnon C, Ridley AJ, Thrasher AJ.

J Immunol. 2001 Apr 15;166(8):4831-4.

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