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Results: 1 to 20 of 90

Similar articles for PubMed (Select 11449000)

1.
2.

Connections between connexins, calcium, and cataracts in the lens.

Gao J, Sun X, Martinez-Wittinghan FJ, Gong X, White TW, Mathias RT.

J Gen Physiol. 2004 Oct;124(4):289-300.

3.
4.

Regional changes of AQP0-dependent square array junction and gap junction associated with cortical cataract formation in the Emory mutant mouse.

Biswas SK, Brako L, Gu S, Jiang JX, Lo WK.

Exp Eye Res. 2014 Oct;127:132-42. doi: 10.1016/j.exer.2014.07.020. Epub 2014 Jul 31.

PMID:
25088353
5.

Dominant cataracts result from incongruous mixing of wild-type lens connexins.

Martinez-Wittinghan FJ, Sellitto C, Li L, Gong X, Brink PR, Mathias RT, White TW.

J Cell Biol. 2003 Jun 9;161(5):969-78. Epub 2003 Jun 2.

6.

Aberrant lens fiber differentiation in anterior subcapsular cataract formation: a process dependent on reduced levels of Pax6.

Lovicu FJ, Steven P, Saika S, McAvoy JW.

Invest Ophthalmol Vis Sci. 2004 Jun;45(6):1946-53.

PMID:
15161862
7.

Neuron glia-related cell adhesion molecule (NrCAM) promotes topographic retinocollicular mapping.

Dai J, Buhusi M, Demyanenko GP, Brennaman LH, Hruska M, Dalva MB, Maness PF.

PLoS One. 2013 Sep 2;8(9):e73000. doi: 10.1371/journal.pone.0073000. eCollection 2013.

8.
9.

Absence of alpha3 (Cx46) and alpha8 (Cx50) connexins leads to cataracts by affecting lens inner fiber cells.

Xia CH, Cheng C, Huang Q, Cheung D, Li L, Dunia I, Benedetti LE, Horwitz J, Gong X.

Exp Eye Res. 2006 Sep;83(3):688-96. Epub 2006 May 11.

PMID:
16696970
10.
11.

Prenatal lens development in connexin43 and connexin50 double knockout mice.

White TW, Sellitto C, Paul DL, Goodenough DA.

Invest Ophthalmol Vis Sci. 2001 Nov;42(12):2916-23.

PMID:
11687537
14.

PKCgamma knockout mouse lenses are more susceptible to oxidative stress damage.

Lin D, Barnett M, Lobell S, Madgwick D, Shanks D, Willard L, Zampighi GA, Takemoto DJ.

J Exp Biol. 2006 Nov;209(Pt 21):4371-8.

15.

A direct interaction of axonin-1 with NgCAM-related cell adhesion molecule (NrCAM) results in guidance, but not growth of commissural axons.

Fitzli D, Stoeckli ET, Kunz S, Siribour K, Rader C, Kunz B, Kozlov SV, Buchstaller A, Lane RP, Suter DM, Dreyer WJ, Sonderegger P.

J Cell Biol. 2000 May 15;149(4):951-68.

16.

Diverse gap junctions modulate distinct mechanisms for fiber cell formation during lens development and cataractogenesis.

Xia CH, Liu H, Cheung D, Cheng C, Wang E, Du X, Beutler B, Lo WK, Gong X.

Development. 2006 May;133(10):2033-40. Epub 2006 Apr 12.

17.

Oxidative stress, lens gap junctions, and cataracts.

Berthoud VM, Beyer EC.

Antioxid Redox Signal. 2009 Feb;11(2):339-53. doi: 10.1089/ars.2008.2119. Review.

18.

Connexin mutation that causes dominant congenital cataracts inhibits gap junctions, but not hemichannels, in a dominant negative manner.

Banks EA, Toloue MM, Shi Q, Zhou ZJ, Liu J, Nicholson BJ, Jiang JX.

J Cell Sci. 2009 Feb 1;122(Pt 3):378-88. doi: 10.1242/jcs.034124. Epub 2009 Jan 6.

19.

Impaired cytoskeletal organization and membrane integrity in lens fibers of a Rho GTPase functional knockout transgenic mouse.

Maddala R, Deng PF, Costello JM, Wawrousek EF, Zigler JS, Rao VP.

Lab Invest. 2004 Jun;84(6):679-92.

20.

NrCAM deletion causes topographic mistargeting of thalamocortical axons to the visual cortex and disrupts visual acuity.

Demyanenko GP, Riday TT, Tran TS, Dalal J, Darnell EP, Brennaman LH, Sakurai T, Grumet M, Philpot BD, Maness PF.

J Neurosci. 2011 Jan 26;31(4):1545-58. doi: 10.1523/JNEUROSCI.4467-10.2011.

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