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Items: 1 to 20 of 102

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Angiotensin II-induced cardiomyocyte hypertrophy and cardiac fibrosis in stroke-prone spontaneously hypertensive rats.

Ikeda Y, Nakamura T, Takano H, Kimura H, Obata JE, Takeda S, Hata A, Shido K, Mochizuki S, Yoshida Y.

J Lab Clin Med. 2000 Apr;135(4):353-9.

PMID:
10779052
6.

Effect of an angiotensin II receptor antagonist, TCV-116, on cardiac hypertrophy and coronary circulation in spontaneously hypertensive rats.

Takeda K, Fujita H, Nakamura K, Uchida A, Tanaka M, Itoh H, Nakata T, Sasaki S, Nakagawa M.

Blood Press Suppl. 1994;5:94-8.

PMID:
7889211
8.

Angiotensin AT1 receptor antagonism and protection against cardiovascular end-organ damage.

Nishikawa K.

J Hum Hypertens. 1998 May;12(5):301-9. Review.

PMID:
9655651
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Protective effects of candesartan cilexetil (TCV-116) against stroke, kidney dysfunction and cardiac hypertrophy in stroke-prone spontaneously hypertensive rats.

Inada Y, Wada T, Ojima M, Sanada T, Shibouta Y, Kanagawa R, Ishimura Y, Fujisawa Y, Nishikawa K.

Clin Exp Hypertens. 1997 Oct;19(7):1079-99.

PMID:
9310205
10.

Combined blockade of AT1-receptors and ACE synergistically potentiates antihypertensive effects in SHR.

Raasch W, Jöhren O, Schwartz S, Gieselberg A, Dominiak P.

J Hypertens. 2004 Mar;22(3):611-8.

PMID:
15076168
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Myocyte hypertrophy and capillarization in spontaneously hypertensive stroke-prone rats.

Koyama T, Gao M, Batra S, Togashi H, Saito H.

Adv Exp Med Biol. 1997;411:365-8.

PMID:
9269450
14.

Three-month effects of candesartan cilexetil, an angiotensin II type 1 (AT1) receptor antagonist, on left ventricular mass and hemodynamics in patients with essential hypertension.

Mitsunami K, Inoue S, Maeda K, Endoh S, Takahashi M, Okada M, Sugihara H, Kinoshita M.

Cardiovasc Drugs Ther. 1998 Oct;12(5):469-74.

PMID:
9926278
15.

[Ameliorative effects on retinal disorder in diabetic SHRSP (stroke-prone spontaneously hypertensive rat)].

Nagisa Y, Shintani A, Nakagawa S.

Nihon Rinsho. 2002 Oct;60(10):1975-80. Review. Japanese.

PMID:
12397694
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Roles of renin-angiotensin and endothelin systems in development of diastolic heart failure in hypertensive hearts.

Yamamoto K, Masuyama T, Sakata Y, Mano T, Nishikawa N, Kondo H, Akehi N, Kuzuya T, Miwa T, Hori M.

Cardiovasc Res. 2000 Aug;47(2):274-83.

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Normalizing the expression of nitric oxide synthase by low-dose AT1 receptor antagonism parallels improved vascular morphology in hypertensive rats.

Bennai F, Morsing P, Paliege A, Ketteler M, Mayer B, Tapp R, Bachmann S.

J Am Soc Nephrol. 1999 Jan;10 Suppl 11:S104-15.

PMID:
9892150
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