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The following terms were not found in PubMed: Gm18629, Gm18629
Page 1
Showing results for ((Gm18629 / snrpn) OR (snrpn / Gm18629)) AND (fusion) AND (mouse)
Your search for ((Gm18629 / Snrpa) OR (Snrpa / Gm18629)) AND (fusion) AND (mouse) retrieved no results
Epigenetic therapy of Prader-Willi syndrome.
Kim Y, Wang SE, Jiang YH. Kim Y, et al. Transl Res. 2019 Jun;208:105-118. doi: 10.1016/j.trsl.2019.02.012. Epub 2019 Mar 5. Transl Res. 2019. PMID: 30904443 Free PMC article. Review.
Inactivation of H3K9m3 methyltransferase SETDB1 and zinc finger protein ZNF274 results in reactivation of SNRPN and SNORD116 cluster from the maternal chromosomes in PWS patient iPSCs and iPSC-derived neurons, respectively. High content screening of small molecule librarie …
Inactivation of H3K9m3 methyltransferase SETDB1 and zinc finger protein ZNF274 results in reactivation of SNRPN and SNORD116 cluster …
Paternal preconceptional diet enriched with n-3 polyunsaturated fatty acids affects offspring brain function in mice.
Li M, Shi Q, Jiang X, Liu X, Han W, Fan X, Li P, Qi K. Li M, et al. Front Nutr. 2022 Oct 28;9:969848. doi: 10.3389/fnut.2022.969848. eCollection 2022. Front Nutr. 2022. PMID: 36386900 Free PMC article.
Then they were mated to 10-week-old virgin female C57BL/6J mice to generate the offspring. The results showed that paternal n-3 PUFA supplementation in preconception reduced the anxiety- and depressive-like behavior, and improved sociability, learning and memory in the off …
Then they were mated to 10-week-old virgin female C57BL/6J mice to generate the offspring. The results showed that paternal n-3 PUFA …
Characterization of the human Snrpn minimal promoter and cis elements within it.
Green Finberg Y, Kantor B, Hershko AY, Razin A. Green Finberg Y, et al. Gene. 2003 Jan 30;304:201-6. doi: 10.1016/s0378-1119(02)01212-x. Gene. 2003. PMID: 12568729
Prader-Willi syndrome (PWS) and Angelman syndrome (AS) are caused by loss of gene function of the imprinted genes including Snrpn within a 2 Mb domain on chromosome 15q11-13. Based on microdeletions in PWS and AS patients, a 4.3 sequence around Snrpn promoter/exon 1 …
Prader-Willi syndrome (PWS) and Angelman syndrome (AS) are caused by loss of gene function of the imprinted genes including Snrpn wit …
Evidence for translational regulation of the imprinted Snurf-Snrpn locus in mice.
Tsai TF, Chen KS, Weber JS, Justice MJ, Beaudet AL. Tsai TF, et al. Hum Mol Genet. 2002 Jul 1;11(14):1659-68. doi: 10.1093/hmg/11.14.1659. Hum Mol Genet. 2002. PMID: 12075010
In studies of genomic imprinting in the Prader-Willi/Angelman domain, an agouti coat color cassette was inserted into the downstream open reading frame (ORF) of the imprinted bicistronic Snurf-Snrpn locus in the mouse. The fusion gene was maternally silenced, …
In studies of genomic imprinting in the Prader-Willi/Angelman domain, an agouti coat color cassette was inserted into the downstream open re …
Deficiency of Rbbp1/Arid4a and Rbbp1l1/Arid4b alters epigenetic modifications and suppresses an imprinting defect in the PWS/AS domain.
Wu MY, Tsai TF, Beaudet AL. Wu MY, et al. Genes Dev. 2006 Oct 15;20(20):2859-70. doi: 10.1101/gad.1452206. Genes Dev. 2006. PMID: 17043311 Free PMC article.
We recovered these two genes from gene trap mutagenesis selecting for altered expression of an Snrpn-EGFP fusion gene strategy. RBBP1/ARID4A is an Rb-binding protein. RBBP1/ARID4A interacts with RBBP1L1/ARID4B and with the Snrpn promoter, implying that both a …
We recovered these two genes from gene trap mutagenesis selecting for altered expression of an Snrpn-EGFP fusion gene strategy …
Early apoptotic reorganization of spliceosomal proteins involves caspases, CAD and rearrangement of NuMA.
Dieker J, Iglesias-Guimarais V, Décossas M, Stevenin J, van der Vlag J, Yuste VJ, Muller S. Dieker J, et al. Traffic. 2012 Feb;13(2):257-72. doi: 10.1111/j.1600-0854.2011.01307.x. Epub 2011 Nov 23. Traffic. 2012. PMID: 22023725 Free article.
Overexpression of CAD in IMR-5 cells results in the dissociation of IGCs from chromatin, but the fusion into a single structure remains disturbed. Nuclear matrix protein NuMA is reorganized in a caspase-dependent way around fused IGCs. ...
Overexpression of CAD in IMR-5 cells results in the dissociation of IGCs from chromatin, but the fusion into a single structure remai …
Multiple protein: protein interactions between the snRNP common core proteins.
Fury MG, Zhang W, Christodoulopoulos I, Zieve GW. Fury MG, et al. Exp Cell Res. 1997 Nov 25;237(1):63-9. doi: 10.1006/excr.1997.3750. Exp Cell Res. 1997. PMID: 9417867
The snRNP core proteins (B, D3, D2, D1, E, F, and G) assemble with snRNA and form the snRNP core particle with a suggested stoichiometry of B2[D1, D2(E, F, G)2]D3. The newly synthesized snRNP core proteins are stored in the …
The snRNP core proteins (B, D3, D2, D1, E, F, and G) assemble with snRNA and form the snRNP core particle …
Specificity and idiotypic analysis of a monoclonal anti-Sm antibody with anti-DNA activity.
Pisetsky DS, Hoch SO, Klatt CL, O'Donnell MA, Keene JD. Pisetsky DS, et al. J Immunol. 1985 Dec;135(6):4080-5. J Immunol. 1985. PMID: 3877763
To investigate the mechanisms of anti-Sm expression in murine systemic lupus erythematosus (SLE), the idiotypic determinants of a monoclonal anti-Sm antibody were studied. This antibody, 2G7, was derived from the fusion of spleen cells of an autoimmune MRL-lpr/lpr mouse …
To investigate the mechanisms of anti-Sm expression in murine systemic lupus erythematosus (SLE), the idiotypic determinants of a monoclonal …
Efficient Cre-loxP-induced mitotic recombination in mouse embryonic stem cells.
Liu P, Jenkins NA, Copeland NG. Liu P, et al. Nat Genet. 2002 Jan;30(1):66-72. doi: 10.1038/ng788. Epub 2001 Dec 10. Nat Genet. 2002. PMID: 11740496
Here we show that mitotic recombination can be reproducibly induced in mouse embryonic stem (ES) cells, by Cre/loxP technology, at frequencies ranging from 4.2 x 10(-5) (Snrpn) to 7.0 x 10(-3) (D7Mit178) for single allelic loxP sites, and to 5.0 x 10(-2) (D7Mit178) …
Here we show that mitotic recombination can be reproducibly induced in mouse embryonic stem (ES) cells, by Cre/loxP technology, at fr …
Residual Cajal bodies in coilin knockout mice fail to recruit Sm snRNPs and SMN, the spinal muscular atrophy gene product.
Tucker KE, Berciano MT, Jacobs EY, LePage DF, Shpargel KB, Rossire JJ, Chan EK, Lafarga M, Conlon RA, Matera AG. Tucker KE, et al. J Cell Biol. 2001 Jul 23;154(2):293-307. doi: 10.1083/jcb.200104083. J Cell Biol. 2001. PMID: 11470819 Free PMC article.
Analysis of tissues and cell lines from mutant animals reveals the presence of extranucleolar foci that contain Fb and Nopp but not other typical nucleolar markers. These so-called "residual" CBs neither condense Sm proteins nor recruit members of the SMN protein complex. …
Analysis of tissues and cell lines from mutant animals reveals the presence of extranucleolar foci that contain Fb and Nopp but not other ty …
19 results