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Items: 1 to 20 of 62


Role of Junctin protein interactions in cellular dynamics of calsequestrin polymer upon calcium perturbation.

Lee KW, Maeng JS, Choi JY, Lee YR, Hwang CY, Park SS, Park HK, Chung BH, Lee SG, Kim YS, Jeon H, Eom SH, Kang C, Kim do H, Kwon KS.

J Biol Chem. 2012 Jan 13;287(3):1679-87. doi: 10.1074/jbc.M111.254045. Epub 2011 Nov 28.


Sequential stages in the age-dependent gradual formation and accumulation of tubular aggregates in fast twitch muscle fibers: SERCA and calsequestrin involvement.

Boncompagni S, Protasi F, Franzini-Armstrong C.

Age (Dordr). 2012 Feb;34(1):27-41. doi: 10.1007/s11357-011-9211-y. Epub 2011 Feb 12.


Reduced gain of excitation-contraction coupling in triadin-null myotubes is mediated by the disruption of FKBP12/RyR1 interaction.

Eltit JM, Szpyt J, Li H, Allen PD, Perez CF.

Cell Calcium. 2011 Feb;49(2):128-35. doi: 10.1016/j.ceca.2011.01.005.


Ablation of skeletal muscle triadin impairs FKBP12/RyR1 channel interactions essential for maintaining resting cytoplasmic Ca2+.

Eltit JM, Feng W, Lopez JR, Padilla IT, Pessah IN, Molinski TF, Fruen BR, Allen PD, Perez CF.

J Biol Chem. 2010 Dec 3;285(49):38453-62. doi: 10.1074/jbc.M110.164525. Epub 2010 Oct 6.


Fiber type composition of the sternomastoid and diaphragm muscles of dystrophin-deficient mdx mice.

Guido AN, Campos GE, Neto HS, Marques MJ, Minatel E.

Anat Rec (Hoboken). 2010 Oct;293(10):1722-8. doi: 10.1002/ar.21224. Epub 2010 Aug 20.


Rough endoplasmic reticulum to junctional sarcoplasmic reticulum trafficking of calsequestrin in adult cardiomyocytes.

McFarland TP, Milstein ML, Cala SE.

J Mol Cell Cardiol. 2010 Oct;49(4):556-64. doi: 10.1016/j.yjmcc.2010.05.012. Epub 2010 Jun 4.


STUDIES OF THE TRIAD : I. Structure of the Junction in Frog Twitch Fibers.

Franzini-Armstrong C.

J Cell Biol. 1970 Nov 1;47(2):488-99.


Triadin deletion induces impaired skeletal muscle function.

Oddoux S, Brocard J, Schweitzer A, Szentesi P, Giannesini B, Brocard J, Fauré J, Pernet-Gallay K, Bendahan D, Lunardi J, Csernoch L, Marty I.

J Biol Chem. 2009 Dec 11;284(50):34918-29. doi: 10.1074/jbc.M109.022442. Epub 2009 Oct 19.


New roles of calsequestrin and triadin in cardiac muscle.

Knollmann BC.

J Physiol. 2009 Jul 1;587(Pt 13):3081-7. doi: 10.1113/jphysiol.2009.172098. Epub 2009 May 18. Review.


Deconstructing calsequestrin. Complex buffering in the calcium store of skeletal muscle.

Royer L, Ríos E.

J Physiol. 2009 Jul 1;587(Pt 13):3101-11. doi: 10.1113/jphysiol.2009.171934. Epub 2009 Apr 29. Review.


Junctin and triadin each activate skeletal ryanodine receptors but junctin alone mediates functional interactions with calsequestrin.

Wei L, Gallant EM, Dulhunty AF, Beard NA.

Int J Biochem Cell Biol. 2009 Nov;41(11):2214-24. doi: 10.1016/j.biocel.2009.04.017. Epub 2009 May 4.


Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmias.

Chopra N, Yang T, Asghari P, Moore ED, Huke S, Akin B, Cattolica RA, Perez CF, Hlaing T, Knollmann-Ritschel BE, Jones LR, Pessah IN, Allen PD, Franzini-Armstrong C, Knollmann BC.

Proc Natl Acad Sci U S A. 2009 May 5;106(18):7636-41. doi: 10.1073/pnas.0902919106. Epub 2009 Apr 21.


Calsequestrin isoforms localize to different ER subcompartments: evidence for polymer and heteropolymer-dependent localization.

Milstein ML, Houle TD, Cala SE.

Exp Cell Res. 2009 Feb 1;315(3):523-34. doi: 10.1016/j.yexcr.2008.11.006. Epub 2008 Nov 25.


Altered expression of triadin 95 causes parallel changes in localized Ca2+ release events and global Ca2+ signals in skeletal muscle cells in culture.

Fodor J, Gönczi M, Sztretye M, Dienes B, Oláh T, Szabó L, Csoma E, Szentesi P, Szigeti GP, Marty I, Csernoch L.

J Physiol. 2008 Dec 1;586(Pt 23):5803-18. doi: 10.1113/jphysiol.2008.160457. Epub 2008 Oct 9.


Altered stored calcium release in skeletal myotubes deficient of triadin and junctin.

Wang Y, Li X, Duan H, Fulton TR, Eu JP, Meissner G.

Cell Calcium. 2009 Jan;45(1):29-37. doi: 10.1016/j.ceca.2008.05.006. Epub 2008 Jul 11.


Structural differentiation of skeletal muscle fibers in the absence of innervation in humans.

Boncompagni S, Kern H, Rossini K, Hofer C, Mayr W, Carraro U, Protasi F.

Proc Natl Acad Sci U S A. 2007 Dec 4;104(49):19339-44. Epub 2007 Nov 27.


Triadins modulate intracellular Ca(2+) homeostasis but are not essential for excitation-contraction coupling in skeletal muscle.

Shen X, Franzini-Armstrong C, Lopez JR, Jones LR, Kobayashi YM, Wang Y, Kerrick WG, Caswell AH, Potter JD, Miller T, Allen PD, Perez CF.

J Biol Chem. 2007 Dec 28;282(52):37864-74. Epub 2007 Nov 2.


Reorganized stores and impaired calcium handling in skeletal muscle of mice lacking calsequestrin-1.

Paolini C, Quarta M, Nori A, Boncompagni S, Canato M, Volpe P, Allen PD, Reggiani C, Protasi F.

J Physiol. 2007 Sep 1;583(Pt 2):767-84. Epub 2007 Jul 12.


Increased Ca2+ storage capacity of the skeletal muscle sarcoplasmic reticulum of transgenic mice over-expressing membrane bound calcium binding protein junctate.

Divet A, Paesante S, Grasso C, Cavagna D, Tiveron C, Paolini C, Protasi F, Huchet-Cadiou C, Treves S, Zorzato F.

J Cell Physiol. 2007 Nov;213(2):464-74.


On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure.

Gergs U, Berndt T, Buskase J, Jones LR, Kirchhefer U, Müller FU, Schlüter KD, Schmitz W, Neumann J.

Am J Physiol Heart Circ Physiol. 2007 Jul;293(1):H728-34. Epub 2007 Mar 30.

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