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Items: 1 to 20 of 102

1.

TWEAK causes myotube atrophy through coordinated activation of ubiquitin-proteasome system, autophagy, and caspases.

Bhatnagar S, Mittal A, Gupta SK, Kumar A.

J Cell Physiol. 2012 Mar;227(3):1042-51. doi: 10.1002/jcp.22821.

2.

Identification of genes that elicit disuse muscle atrophy via the transcription factors p50 and Bcl-3.

Wu CL, Kandarian SC, Jackman RW.

PLoS One. 2011 Jan 13;6(1):e16171. doi: 10.1371/journal.pone.0016171.

3.

Exercise increases mitochondrial PGC-1alpha content and promotes nuclear-mitochondrial cross-talk to coordinate mitochondrial biogenesis.

Safdar A, Little JP, Stokl AJ, Hettinga BP, Akhtar M, Tarnopolsky MA.

J Biol Chem. 2011 Mar 25;286(12):10605-17. doi: 10.1074/jbc.M110.211466. Epub 2011 Jan 18.

4.

Targeted ablation of TRAF6 inhibits skeletal muscle wasting in mice.

Paul PK, Gupta SK, Bhatnagar S, Panguluri SK, Darnay BG, Choi Y, Kumar A.

J Cell Biol. 2010 Dec 27;191(7):1395-411. doi: 10.1083/jcb.201006098.

5.

Tumor necrosis factor-α regulates distinct molecular pathways and gene networks in cultured skeletal muscle cells.

Bhatnagar S, Panguluri SK, Gupta SK, Dahiya S, Lundy RF, Kumar A.

PLoS One. 2010 Oct 12;5(10):e13262. doi: 10.1371/journal.pone.0013262.

6.

Genetic ablation of TWEAK augments regeneration and post-injury growth of skeletal muscle in mice.

Mittal A, Bhatnagar S, Kumar A, Paul PK, Kuang S, Kumar A.

Am J Pathol. 2010 Oct;177(4):1732-42. doi: 10.2353/ajpath.2010.100335. Epub 2010 Aug 19.

7.

Peroxisome proliferator-activated receptor gamma coactivator 1alpha or 1beta overexpression inhibits muscle protein degradation, induction of ubiquitin ligases, and disuse atrophy.

Brault JJ, Jespersen JG, Goldberg AL.

J Biol Chem. 2010 Jun 18;285(25):19460-71. doi: 10.1074/jbc.M110.113092. Epub 2010 Apr 19.

8.

Mitochondrial fission and remodelling contributes to muscle atrophy.

Romanello V, Guadagnin E, Gomes L, Roder I, Sandri C, Petersen Y, Milan G, Masiero E, Del Piccolo P, Foretz M, Scorrano L, Rudolf R, Sandri M.

EMBO J. 2010 May 19;29(10):1774-85. doi: 10.1038/emboj.2010.60. Epub 2010 Apr 16.

9.

Signaling pathways perturbing muscle mass.

Glass DJ.

Curr Opin Clin Nutr Metab Care. 2010 May;13(3):225-9. Review.

PMID:
20397318
10.

The TWEAK-Fn14 system is a critical regulator of denervation-induced skeletal muscle atrophy in mice.

Mittal A, Bhatnagar S, Kumar A, Lach-Trifilieff E, Wauters S, Li H, Makonchuk DY, Glass DJ, Kumar A.

J Cell Biol. 2010 Mar 22;188(6):833-49. doi: 10.1083/jcb.200909117.

11.

Regulatory interactions between muscle and the immune system during muscle regeneration.

Tidball JG, Villalta SA.

Am J Physiol Regul Integr Comp Physiol. 2010 May;298(5):R1173-87. doi: 10.1152/ajpregu.00735.2009. Epub 2010 Mar 10. Review.

12.

Genomic profiling of messenger RNAs and microRNAs reveals potential mechanisms of TWEAK-induced skeletal muscle wasting in mice.

Panguluri SK, Bhatnagar S, Kumar A, McCarthy JJ, Srivastava AK, Cooper NG, Lundy RF, Kumar A.

PLoS One. 2010 Jan 19;5(1):e8760. doi: 10.1371/journal.pone.0008760.

13.

TNF-alpha-mediated reduction in PGC-1alpha may impair skeletal muscle function after cigarette smoke exposure.

Tang K, Wagner PD, Breen EC.

J Cell Physiol. 2010 Feb;222(2):320-7. doi: 10.1002/jcp.21955.

PMID:
19859910
14.

p38 Mitogen-activated protein kinase modulates endotoxin-induced diaphragm caspase activation.

Supinski GS, Ji XY, Callahan LA.

Am J Respir Cell Mol Biol. 2010 Jul;43(1):121-7. doi: 10.1165/rcmb.2008-0395OC. Epub 2009 Aug 28.

15.

Interleukin-1 stimulates catabolism in C2C12 myotubes.

Li W, Moylan JS, Chambers MA, Smith J, Reid MB.

Am J Physiol Cell Physiol. 2009 Sep;297(3):C706-14. doi: 10.1152/ajpcell.00626.2008. Epub 2009 Jul 22.

16.

The JNK MAP kinase pathway contributes to the development of endotoxin-induced diaphragm caspase activation.

Supinski GS, Ji X, Callahan LA.

Am J Physiol Regul Integr Comp Physiol. 2009 Sep;297(3):R825-34. doi: 10.1152/ajpregu.90849.2008. Epub 2009 Jul 15.

17.

During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylation.

Cohen S, Brault JJ, Gygi SP, Glass DJ, Valenzuela DM, Gartner C, Latres E, Goldberg AL.

J Cell Biol. 2009 Jun 15;185(6):1083-95. doi: 10.1083/jcb.200901052. Epub 2009 Jun 8.

18.

A novel role for tumor necrosis factor-like weak inducer of apoptosis (TWEAK) in the development of cardiac dysfunction and failure.

Jain M, Jakubowski A, Cui L, Shi J, Su L, Bauer M, Guan J, Lim CC, Naito Y, Thompson JS, Sam F, Ambrose C, Parr M, Crowell T, Lincecum JM, Wang MZ, Hsu YM, Zheng TS, Michaelson JS, Liao R, Burkly LC.

Circulation. 2009 Apr 21;119(15):2058-68. doi: 10.1161/CIRCULATIONAHA.108.837286. Epub 2009 Apr 6.

19.

TNF-like weak inducer of apoptosis (TWEAK) activates proinflammatory signaling pathways and gene expression through the activation of TGF-beta-activated kinase 1.

Kumar M, Makonchuk DY, Li H, Mittal A, Kumar A.

J Immunol. 2009 Feb 15;182(4):2439-48. doi: 10.4049/jimmunol.0803357.

20.

IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.

Zhang L, Du J, Hu Z, Han G, Delafontaine P, Garcia G, Mitch WE.

J Am Soc Nephrol. 2009 Mar;20(3):604-12. doi: 10.1681/ASN.2008060628. Epub 2009 Jan 21.

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