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Results: 1 to 20 of 41

References for PMC Articles for PubMed (Select 20618070)

1.

Redox signaling (cross-talk) from and to mitochondria involves mitochondrial pores and reactive oxygen species.

Daiber A.

Biochim Biophys Acta. 2010 Jun-Jul;1797(6-7):897-906. doi: 10.1016/j.bbabio.2010.01.032. Epub 2010 Feb 1. Review.

2.

Lack of oxygen deactivates mitochondrial complex I: implications for ischemic injury?

Galkin A, Abramov AY, Frakich N, Duchen MR, Moncada S.

J Biol Chem. 2009 Dec 25;284(52):36055-61. doi: 10.1074/jbc.M109.054346. Epub 2009 Oct 27.

3.

Targeting and regulation of reactive oxygen species generation by Nox family NADPH oxidases.

Leto TL, Morand S, Hurt D, Ueyama T.

Antioxid Redox Signal. 2009 Oct;11(10):2607-19. doi: 10.1089/ARS.2009.2637. Review.

4.

NADPH oxidase is required for the sensory plasticity of the carotid body by chronic intermittent hypoxia.

Peng YJ, Nanduri J, Yuan G, Wang N, Deneris E, Pendyala S, Natarajan V, Kumar GK, Prabhakar NR.

J Neurosci. 2009 Apr 15;29(15):4903-10. doi: 10.1523/JNEUROSCI.4768-08.2009.

5.

Neonatal intermittent hypoxia leads to long-lasting facilitation of acute hypoxia-evoked catecholamine secretion from rat chromaffin cells.

Souvannakitti D, Kumar GK, Fox A, Prabhakar NR.

J Neurophysiol. 2009 Jun;101(6):2837-46. doi: 10.1152/jn.00036.2009. Epub 2009 Apr 1.

6.
7.

NADPH oxidase activity is necessary for acute intermittent hypoxia-induced phrenic long-term facilitation.

MacFarlane PM, Satriotomo I, Windelborn JA, Mitchell GS.

J Physiol. 2009 May 1;587(Pt 9):1931-42. doi: 10.1113/jphysiol.2008.165597. Epub 2009 Feb 23. Review.

8.

How mitochondria produce reactive oxygen species.

Murphy MP.

Biochem J. 2009 Jan 1;417(1):1-13. doi: 10.1042/BJ20081386. Review.

9.

Challenges in elucidating structure and mechanism of proton pumping NADH:ubiquinone oxidoreductase (complex I).

Zickermann V, Dröse S, Tocilescu MA, Zwicker K, Kerscher S, Brandt U.

J Bioenerg Biomembr. 2008 Oct;40(5):475-83. doi: 10.1007/s10863-008-9171-9. Epub 2008 Nov 4. Review.

PMID:
18982432
10.

Induction of HIF-1alpha expression by intermittent hypoxia: involvement of NADPH oxidase, Ca2+ signaling, prolyl hydroxylases, and mTOR.

Yuan G, Nanduri J, Khan S, Semenza GL, Prabhakar NR.

J Cell Physiol. 2008 Dec;217(3):674-85. doi: 10.1002/jcp.21537.

11.

Hypoxia activates NADPH oxidase to increase [ROS]i and [Ca2+]i through the mitochondrial ROS-PKCepsilon signaling axis in pulmonary artery smooth muscle cells.

Rathore R, Zheng YM, Niu CF, Liu QH, Korde A, Ho YS, Wang YX.

Free Radic Biol Med. 2008 Nov 1;45(9):1223-31. doi: 10.1016/j.freeradbiomed.2008.06.012. Epub 2008 Jun 21.

12.

Complex I within oxidatively stressed bovine heart mitochondria is glutathionylated on Cys-531 and Cys-704 of the 75-kDa subunit: potential role of CYS residues in decreasing oxidative damage.

Hurd TR, Requejo R, Filipovska A, Brown S, Prime TA, Robinson AJ, Fearnley IM, Murphy MP.

J Biol Chem. 2008 Sep 5;283(36):24801-15. doi: 10.1074/jbc.M803432200. Epub 2008 Jul 8.

13.

First evidence for a crosstalk between mitochondrial and NADPH oxidase-derived reactive oxygen species in nitroglycerin-triggered vascular dysfunction.

Wenzel P, Mollnau H, Oelze M, Schulz E, Wickramanayake JM, Müller J, Schuhmacher S, Hortmann M, Baldus S, Gori T, Brandes RP, Münzel T, Daiber A.

Antioxid Redox Signal. 2008 Aug;10(8):1435-47. doi: 10.1089/ars.2007.1969.

PMID:
18522491
14.
15.

S-nitrosation of mitochondrial complex I depends on its structural conformation.

Galkin A, Moncada S.

J Biol Chem. 2007 Dec 28;282(52):37448-53. Epub 2007 Oct 23.

16.

Reactive oxygen species contribute to sleep apnea-induced hypertension in rats.

Troncoso Brindeiro CM, da Silva AQ, Allahdadi KJ, Youngblood V, Kanagy NL.

Am J Physiol Heart Circ Physiol. 2007 Nov;293(5):H2971-6. Epub 2007 Aug 31.

17.

Increased secretory capacity of mouse adrenal chromaffin cells by chronic intermittent hypoxia: involvement of protein kinase C.

Kuri BA, Khan SA, Chan SA, Prabhakar NR, Smith CB.

J Physiol. 2007 Oct 1;584(Pt 1):313-9. Epub 2007 Aug 16.

18.

ROS signaling in systemic and cellular responses to chronic intermittent hypoxia.

Prabhakar NR, Kumar GK, Nanduri J, Semenza GL.

Antioxid Redox Signal. 2007 Sep;9(9):1397-403. Review.

PMID:
17627465
19.

Preconditioning and postconditioning: the essential role of the mitochondrial permeability transition pore.

Lim SY, Davidson SM, Hausenloy DJ, Yellon DM.

Cardiovasc Res. 2007 Aug 1;75(3):530-5. Epub 2007 May 4.

20.

Altered carotid body function by intermittent hypoxia in neonates and adults: relevance to recurrent apneas.

Prabhakar NR, Peng YJ, Kumar GK, Pawar A.

Respir Physiol Neurobiol. 2007 Jul 1;157(1):148-53. Epub 2007 Jan 11. Review.

PMID:
17317339
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