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Items: 1 to 20 of 25

1.

FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis.

Shimada T, Hasegawa H, Yamazaki Y, Muto T, Hino R, Takeuchi Y, Fujita T, Nakahara K, Fukumoto S, Yamashita T.

J Bone Miner Res. 2004 Mar;19(3):429-35. Epub 2003 Dec 29.

2.

FGF-23 in fibrous dysplasia of bone and its relationship to renal phosphate wasting.

Riminucci M, Collins MT, Fedarko NS, Cherman N, Corsi A, White KE, Waguespack S, Gupta A, Hannon T, Econs MJ, Bianco P, Gehron Robey P.

J Clin Invest. 2003 Sep;112(5):683-92.

3.

Fibroblast growth factor 23 in oncogenic osteomalacia and X-linked hypophosphatemia.

Jonsson KB, Zahradnik R, Larsson T, White KE, Sugimoto T, Imanishi Y, Yamamoto T, Hampson G, Koshiyama H, Ljunggren O, Oba K, Yang IM, Miyauchi A, Econs MJ, Lavigne J, Jüppner H.

N Engl J Med. 2003 Apr 24;348(17):1656-63.

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5.

Increased circulatory level of biologically active full-length FGF-23 in patients with hypophosphatemic rickets/osteomalacia.

Yamazaki Y, Okazaki R, Shibata M, Hasegawa Y, Satoh K, Tajima T, Takeuchi Y, Fujita T, Nakahara K, Yamashita T, Fukumoto S.

J Clin Endocrinol Metab. 2002 Nov;87(11):4957-60.

PMID:
12414858
6.

Mutant FGF-23 responsible for autosomal dominant hypophosphatemic rickets is resistant to proteolytic cleavage and causes hypophosphatemia in vivo.

Shimada T, Muto T, Urakawa I, Yoneya T, Yamazaki Y, Okawa K, Takeuchi Y, Fujita T, Fukumoto S, Yamashita T.

Endocrinology. 2002 Aug;143(8):3179-82.

PMID:
12130585
7.

Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23.

White KE, Carn G, Lorenz-Depiereux B, Benet-Pages A, Strom TM, Econs MJ.

Kidney Int. 2001 Dec;60(6):2079-86.

8.

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia.

Shimada T, Mizutani S, Muto T, Yoneya T, Hino R, Takeda S, Takeuchi Y, Fujita T, Fukumoto S, Yamashita T.

Proc Natl Acad Sci U S A. 2001 May 22;98(11):6500-5. Epub 2001 May 8.

9.

The autosomal dominant hypophosphatemic rickets (ADHR) gene is a secreted polypeptide overexpressed by tumors that cause phosphate wasting.

White KE, Jonsson KB, Carn G, Hampson G, Spector TD, Mannstadt M, Lorenz-Depiereux B, Miyauchi A, Yang IM, Ljunggren O, Meitinger T, Strom TM, Jüppner H, Econs MJ.

J Clin Endocrinol Metab. 2001 Feb;86(2):497-500.

PMID:
11157998
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12.

Deficient mineralization of intramembranous bone in vitamin D-24-hydroxylase-ablated mice is due to elevated 1,25-dihydroxyvitamin D and not to the absence of 24,25-dihydroxyvitamin D.

St-Arnaud R, Arabian A, Travers R, Barletta F, Raval-Pandya M, Chapin K, Depovere J, Mathieu C, Christakos S, Demay MB, Glorieux FH.

Endocrinology. 2000 Jul;141(7):2658-66.

PMID:
10875271
13.

PHEX gene and hypophosphatemia.

Drezner MK.

Kidney Int. 2000 Jan;57(1):9-18. Review.

14.

Regulation of small intestinal Na-P(i) type IIb cotransporter by dietary phosphate intake.

Hattenhauer O, Traebert M, Murer H, Biber J.

Am J Physiol. 1999 Oct;277(4 Pt 1):G756-62.

15.

Vitamin D.

Brown AJ, Dusso A, Slatopolsky E.

Am J Physiol. 1999 Aug;277(2 Pt 2):F157-75. Review.

16.

Targeted inactivation of Npt2 in mice leads to severe renal phosphate wasting, hypercalciuria, and skeletal abnormalities.

Beck L, Karaplis AC, Amizuka N, Hewson AS, Ozawa H, Tenenhouse HS.

Proc Natl Acad Sci U S A. 1998 Apr 28;95(9):5372-7.

17.

25-Hydroxyvitamin D3 1alpha-hydroxylase and vitamin D synthesis.

Takeyama K, Kitanaka S, Sato T, Kobori M, Yanagisawa J, Kato S.

Science. 1997 Sep 19;277(5333):1827-30.

18.

A molecular view of proximal tubular inorganic phosphate (Pi) reabsorption and of its regulation.

Murer H, Biber J.

Pflugers Arch. 1997 Feb;433(4):379-89. Review.

PMID:
9000416
19.

Bone histomorphometry in hypoparathyroid patients treated with vitamin D.

Langdahl BL, Mortensen L, Vesterby A, Eriksen EF, Charles P.

Bone. 1996 Feb;18(2):103-8.

PMID:
8833203
20.

Renal brush border membrane Na/Pi-cotransport: molecular aspects in PTH-dependent and dietary regulation.

Murer H, Lötscher M, Kaissling B, Levi M, Kempson SA, Biber J.

Kidney Int. 1996 Jun;49(6):1769-73. Review.

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