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Results: 1 to 20 of 81

Similar articles for PubMed (Select 9620684)

1.

CD19 as a membrane-anchored adaptor protein of B lymphocytes: costimulation of lipid and protein kinases by recruitment of Vav.

O'Rourke LM, Tooze R, Turner M, Sandoval DM, Carter RH, Tybulewicz VL, Fearon DT.

Immunity. 1998 May;8(5):635-45.

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CD19 amplifies B lymphocyte signal transduction by regulating Src-family protein tyrosine kinase activation.

Fujimoto M, Poe JC, Jansen PJ, Sato S, Tedder TF.

J Immunol. 1999 Jun 15;162(12):7088-94.

7.

Role of CD19 tyrosine 391 in synergistic activation of B lymphocytes by coligation of CD19 and membrane Ig.

Li X, Sandoval D, Freeberg L, Carter RH.

J Immunol. 1997 Jun 15;158(12):5649-57.

PMID:
9190913
8.

CD46/CD3 costimulation induces morphological changes of human T cells and activation of Vav, Rac, and extracellular signal-regulated kinase mitogen-activated protein kinase.

Zaffran Y, Destaing O, Roux A, Ory S, Nheu T, Jurdic P, Rabourdin-Combe C, Astier AL.

J Immunol. 2001 Dec 15;167(12):6780-5.

9.

Cbl-b, a member of the Sli-1/c-Cbl protein family, inhibits Vav-mediated c-Jun N-terminal kinase activation.

Bustelo XR, Crespo P, López-Barahona M, Gutkind JS, Barbacid M.

Oncogene. 1997 Nov 20;15(21):2511-20.

PMID:
9399639
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11.

Tyrosine phosphorylation of Vav stimulates IL-6 production in mast cells by a Rac/c-Jun N-terminal kinase-dependent pathway.

Song JS, Haleem-Smith H, Arudchandran R, Gomez J, Scott PM, Mill JF, Tan TH, Rivera J.

J Immunol. 1999 Jul 15;163(2):802-10.

13.

Rac-1 dependent stimulation of the JNK/SAPK signaling pathway by Vav.

Crespo P, Bustelo XR, Aaronson DS, Coso OA, Lopez-Barahona M, Barbacid M, Gutkind JS.

Oncogene. 1996 Aug 1;13(3):455-60.

PMID:
8760286
14.

CD19 regulates innate immunity by the toll-like receptor RP105 signaling in B lymphocytes.

Yazawa N, Fujimoto M, Sato S, Miyake K, Asano N, Nagai Y, Takeuchi O, Takeda K, Okochi H, Akira S, Tedder TF, Tamaki K.

Blood. 2003 Aug 15;102(4):1374-80. Epub 2003 Apr 24.

15.

Vav family proteins couple to diverse cell surface receptors.

Moores SL, Selfors LM, Fredericks J, Breit T, Fujikawa K, Alt FW, Brugge JS, Swat W.

Mol Cell Biol. 2000 Sep;20(17):6364-73.

16.

Vav1/2/3-null mice define an essential role for Vav family proteins in lymphocyte development and activation but a differential requirement in MAPK signaling in T and B cells.

Fujikawa K, Miletic AV, Alt FW, Faccio R, Brown T, Hoog J, Fredericks J, Nishi S, Mildiner S, Moores SL, Brugge J, Rosen FS, Swat W.

J Exp Med. 2003 Nov 17;198(10):1595-608.

17.

Vav transformation requires activation of multiple GTPases and regulation of gene expression.

Palmby TR, Abe K, Karnoub AE, Der CJ.

Mol Cancer Res. 2004 Dec;2(12):702-11.

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Defective CD19-dependent signaling in B-1a and B-1b B lymphocyte subpopulations.

Sen G, Wu HJ, Bikah G, Venkataraman C, Robertson DA, Snow EC, Bondada S.

Mol Immunol. 2002 Sep;39(1-2):57-68.

PMID:
12213328
20.

Pleiotropic defects in TCR signaling in a Vav-1-null Jurkat T-cell line.

Cao Y, Janssen EM, Duncan AW, Altman A, Billadeau DD, Abraham RT.

EMBO J. 2002 Sep 16;21(18):4809-19.

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