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Items: 1 to 20 of 87


Nuclear localization of the mitochondrial factor HIGD1A during metabolic stress.

Ameri K, Rajah AM, Nguyen V, Sanders TA, Jahangiri A, Delay M, Donne M, Choi HJ, Tormos KV, Yeghiazarians Y, Jeffrey SS, Rinaudo PF, Rowitch DH, Aghi M, Maltepe E.

PLoS One. 2013 Apr 30;8(4):e62758. doi: 10.1371/journal.pone.0062758. Print 2013.


Regulation of stress-induced nuclear protein redistribution: a new function of Bax and Bak uncoupled from Bcl-x(L).

Lindenboim L, Blacher E, Borner C, Stein R.

Cell Death Differ. 2010 Feb;17(2):346-59. doi: 10.1038/cdd.2009.145. Epub 2009 Oct 9.


Sequential activation of poly(ADP-ribose) polymerase 1, calpains, and Bax is essential in apoptosis-inducing factor-mediated programmed necrosis.

Moubarak RS, Yuste VJ, Artus C, Bouharrour A, Greer PA, Menissier-de Murcia J, Susin SA.

Mol Cell Biol. 2007 Jul;27(13):4844-62. Epub 2007 Apr 30.


Non-canonical function of Bax in stress-induced nuclear protein redistribution.

Lindenboim L, Ferrando-May E, Borner C, Stein R.

Cell Mol Life Sci. 2013 Aug;70(16):3013-27. doi: 10.1007/s00018-013-1306-4. Epub 2013 Mar 9.


GRAMD4 mimics p53 and mediates the apoptotic function of p73 at mitochondria.

John K, Alla V, Meier C, Pützer BM.

Cell Death Differ. 2011 May;18(5):874-86. doi: 10.1038/cdd.2010.153. Epub 2010 Dec 3. Erratum in: Cell Death Differ. 2012 Mar;19(3):552.


HIGD1A Regulates Oxygen Consumption, ROS Production, and AMPK Activity during Glucose Deprivation to Modulate Cell Survival and Tumor Growth.

Ameri K, Jahangiri A, Rajah AM, Tormos KV, Nagarajan R, Pekmezci M, Nguyen V, Wheeler ML, Murphy MP, Sanders TA, Jeffrey SS, Yeghiazarians Y, Rinaudo PF, Costello JF, Aghi MK, Maltepe E.

Cell Rep. 2015 Feb 12. pii: S2211-1247(15)00033-9. doi: 10.1016/j.celrep.2015.01.020. [Epub ahead of print]


DNA-dependent protein kinase is involved in heat shock protein-mediated accumulation of hypoxia-inducible factor-1alpha in hypoxic preconditioned HepG2 cells.

Kang MJ, Jung SM, Kim MJ, Bae JH, Kim HB, Kim JY, Park SJ, Song HS, Kim DW, Kang CD, Kim SH.

FEBS J. 2008 Dec;275(23):5969-81. doi: 10.1111/j.1742-4658.2008.06725.x.


Disruption of hypoxia-inducible transcription factor-prolyl hydroxylase domain-1 (PHD-1-/-) attenuates ex vivo myocardial ischemia/reperfusion injury through hypoxia-inducible factor-1α transcription factor and its target genes in mice.

Adluri RS, Thirunavukkarasu M, Dunna NR, Zhan L, Oriowo B, Takeda K, Sanchez JA, Otani H, Maulik G, Fong GH, Maulik N.

Antioxid Redox Signal. 2011 Oct 1;15(7):1789-97. doi: 10.1089/ars.2010.3769. Epub 2011 Apr 8.


Critical role for mitochondrial oxidative phosphorylation in the activation of tumor suppressors Bax and Bak.

Tomiyama A, Serizawa S, Tachibana K, Sakurada K, Samejima H, Kuchino Y, Kitanaka C.

J Natl Cancer Inst. 2006 Oct 18;98(20):1462-73.


Bnip3 mediates mitochondrial dysfunction and cell death through Bax and Bak.

Kubli DA, Ycaza JE, Gustafsson AB.

Biochem J. 2007 Aug 1;405(3):407-15.


Hypoxia-mediated down-regulation of Bid and Bax in tumors occurs via hypoxia-inducible factor 1-dependent and -independent mechanisms and contributes to drug resistance.

Erler JT, Cawthorne CJ, Williams KJ, Koritzinsky M, Wouters BG, Wilson C, Miller C, Demonacos C, Stratford IJ, Dive C.

Mol Cell Biol. 2004 Apr;24(7):2875-89.


The survival effect of mitochondrial Higd-1a is associated with suppression of cytochrome C release and prevention of caspase activation.

An HJ, Shin H, Jo SG, Kim YJ, Lee JO, Paik SG, Lee H.

Biochim Biophys Acta. 2011 Dec;1813(12):2088-98. doi: 10.1016/j.bbamcr.2011.07.017. Epub 2011 Aug 10.


Distinct hsp70 domains mediate apoptosis-inducing factor release and nuclear accumulation.

Ruchalski K, Mao H, Li Z, Wang Z, Gillers S, Wang Y, Mosser DD, Gabai V, Schwartz JH, Borkan SC.

J Biol Chem. 2006 Mar 24;281(12):7873-80. Epub 2006 Jan 4.


β-lapachone significantly increases the effect of ionizing radiation to cause mitochondrial apoptosis via JNK activation in cancer cells.

Park MT, Song MJ, Lee H, Oh ET, Choi BH, Jeong SY, Choi EK, Park HJ.

PLoS One. 2011;6(10):e25976. doi: 10.1371/journal.pone.0025976. Epub 2011 Oct 6.


Bax phosphorylation association with nucleus and oligomerization after neonatal hypoxia-ischemia.

Infante SK, Oberhauser AF, Perez-Polo JR.

J Neurosci Res. 2013 Sep;91(9):1152-64. doi: 10.1002/jnr.23224. Epub 2013 Jul 3.


Cyclophilin A participates in the nuclear translocation of apoptosis-inducing factor in neurons after cerebral hypoxia-ischemia.

Zhu C, Wang X, Deinum J, Huang Z, Gao J, Modjtahedi N, Neagu MR, Nilsson M, Eriksson PS, Hagberg H, Luban J, Kroemer G, Blomgren K.

J Exp Med. 2007 Aug 6;204(8):1741-8. Epub 2007 Jul 16.


TOM-independent complex formation of Bax and Bak in mammalian mitochondria during TNFalpha-induced apoptosis.

Ross K, Rudel T, Kozjak-Pavlovic V.

Cell Death Differ. 2009 May;16(5):697-707. doi: 10.1038/cdd.2008.194. Epub 2009 Jan 23.


Mechanism of HIF-1alpha-dependent suppression of hypoxia-induced apoptosis in squamous cell carcinoma cells.

Sasabe E, Tatemoto Y, Li D, Yamamoto T, Osaki T.

Cancer Sci. 2005 Jul;96(7):394-402.


Hsp70 overexpression sequesters AIF and reduces neonatal hypoxic/ischemic brain injury.

Matsumori Y, Hong SM, Aoyama K, Fan Y, Kayama T, Sheldon RA, Vexler ZS, Ferriero DM, Weinstein PR, Liu J.

J Cereb Blood Flow Metab. 2005 Jul;25(7):899-910.

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