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Results: 1 to 20 of 152

1.

Chronic occupational exposure to arsenic induces carcinogenic gene signaling networks and neoplastic transformation in human lung epithelial cells.

Stueckle TA, Lu Y, Davis ME, Wang L, Jiang BH, Holaskova I, Schafer R, Barnett JB, Rojanasakul Y.

Toxicol Appl Pharmacol. 2012 Jun 1;261(2):204-16. doi: 10.1016/j.taap.2012.04.003. Epub 2012 Apr 13.

PMID:
22521957
[PubMed - indexed for MEDLINE]
Free PMC Article
2.

Aberrant overexpression of FOXM1 transcription factor plays a critical role in lung carcinogenesis induced by low doses of arsenic.

Liu Y, Hock JM, Van Beneden RJ, Li X.

Mol Carcinog. 2014 May;53(5):380-91. doi: 10.1002/mc.21989. Epub 2012 Dec 19.

PMID:
23255470
[PubMed - indexed for MEDLINE]
3.

Autophagy is a cell self-protective mechanism against arsenic-induced cell transformation.

Zhang T, Qi Y, Liao M, Xu M, Bower KA, Frank JA, Shen HM, Luo J, Shi X, Chen G.

Toxicol Sci. 2012 Dec;130(2):298-308. doi: 10.1093/toxsci/kfs240. Epub 2012 Aug 5.

PMID:
22869613
[PubMed - indexed for MEDLINE]
Free Article
4.

Comparison of gene expression profiles in chromate transformed BEAS-2B cells.

Sun H, Clancy HA, Kluz T, Zavadil J, Costa M.

PLoS One. 2011 Mar 18;6(3):e17982. doi: 10.1371/journal.pone.0017982.

PMID:
21437242
[PubMed - indexed for MEDLINE]
Free PMC Article
5.

Genome-wide analysis of BEAS-2B cells exposed to trivalent arsenicals and dimethylthioarsinic acid.

Chilakapati J, Wallace K, Ren H, Fricke M, Bailey K, Ward W, Creed J, Kitchin K.

Toxicology. 2010 Jan 31;268(1-2):31-9. doi: 10.1016/j.tox.2009.11.018. Epub 2009 Nov 27.

PMID:
19945496
[PubMed - indexed for MEDLINE]
6.

Macrophages facilitate coal tar pitch extract-induced tumorigenic transformation of human bronchial epithelial cells mediated by NF-κB.

Feng F, Wu Y, Zhang S, Liu Y, Qin L, Wu Y, Yan Z, Wu W.

PLoS One. 2012;7(12):e51690. doi: 10.1371/journal.pone.0051690. Epub 2012 Dec 5.

PMID:
23227270
[PubMed - indexed for MEDLINE]
Free PMC Article
7.

Anthraquinones sensitize tumor cells to arsenic cytotoxicity in vitro and in vivo via reactive oxygen species-mediated dual regulation of apoptosis.

Yang J, Li H, Chen YY, Wang XJ, Shi GY, Hu QS, Kang XL, Lu Y, Tang XM, Guo QS, Yi J.

Free Radic Biol Med. 2004 Dec 15;37(12):2027-41.

PMID:
15544921
[PubMed - indexed for MEDLINE]
8.

Chronic exposure to arsenic causes increased cell survival, DNA damage, and increased expression of mitochondrial transcription factor A (mtTFA) in human prostate epithelial cells.

Singh KP, Kumari R, Treas J, DuMond JW.

Chem Res Toxicol. 2011 Mar 21;24(3):340-9. doi: 10.1021/tx1003112. Epub 2011 Jan 14.

PMID:
21235219
[PubMed - indexed for MEDLINE]
9.

Arsenic-induced changes in the gene expression of lung epithelial L2 cells: implications in carcinogenesis.

Posey T, Weng T, Chen Z, Chintagari NR, Wang P, Jin N, Stricker H, Liu L.

BMC Genomics. 2008 Mar 3;9:115. doi: 10.1186/1471-2164-9-115.

PMID:
18315880
[PubMed - indexed for MEDLINE]
Free PMC Article
10.

Reduced reactive oxygen species-generating capacity contributes to the enhanced cell growth of arsenic-transformed epithelial cells.

Chang Q, Pan J, Wang X, Zhang Z, Chen F, Shi X.

Cancer Res. 2010 Jun 15;70(12):5127-35. doi: 10.1158/0008-5472.CAN-10-0007. Epub 2010 Jun 1.

PMID:
20516118
[PubMed - indexed for MEDLINE]
Free PMC Article
11.

An approach to elucidate potential mechanism of renal toxicity of arsenic trioxide.

Sasaki A, Oshima Y, Fujimura A.

Exp Hematol. 2007 Feb;35(2):252-62.

PMID:
17258074
[PubMed - indexed for MEDLINE]
13.

Arsenic modulates heme oxygenase-1, interleukin-6, and vascular endothelial growth factor expression in endothelial cells: roles of ROS, NF-κB, and MAPK pathways.

Wang L, Kou MC, Weng CY, Hu LW, Wang YJ, Wu MJ.

Arch Toxicol. 2012 Jun;86(6):879-96. doi: 10.1007/s00204-012-0845-z. Epub 2012 Apr 10.

PMID:
22488045
[PubMed - indexed for MEDLINE]
14.

Arsenic trioxide (As2O3) reduces the invasive and metastatic properties of cervical cancer cells in vitro and in vivo.

Yu J, Qian H, Li Y, Wang Y, Zhang X, Liang X, Fu M, Lin C.

Gynecol Oncol. 2007 Aug;106(2):400-6. Epub 2007 May 21.

PMID:
17512576
[PubMed - indexed for MEDLINE]
15.

Microarray analysis revealed dysregulation of multiple genes associated with chemoresistance to As(2)O(3) and increased tumor aggressiveness in a newly established arsenic-resistant ovarian cancer cell line, OVCAR-3/AsR.

Ong PS, Chan SY, Ho PC.

Eur J Pharm Sci. 2012 Feb 14;45(3):367-78. doi: 10.1016/j.ejps.2011.12.003. Epub 2011 Dec 9.

PMID:
22178533
[PubMed - indexed for MEDLINE]
16.

Association of c-myc overexpression and hyperproliferation with arsenite-induced malignant transformation.

Chen H, Liu J, Zhao CQ, Diwan BA, Merrick BA, Waalkes MP.

Toxicol Appl Pharmacol. 2001 Sep 15;175(3):260-8.

PMID:
11559025
[PubMed - indexed for MEDLINE]
17.

Indirect effects of Bax and Bak initiate the mitochondrial alterations that lead to cytochrome c release during arsenic trioxide-induced apoptosis.

Nutt LK, Gogvadze V, Uthaisang W, Mirnikjoo B, McConkey DJ, Orrenius S.

Cancer Biol Ther. 2005 Apr;4(4):459-67. Epub 2005 Apr 28.

PMID:
15846091
[PubMed - indexed for MEDLINE]
Free Article
18.

Multifunctional role of Bcl-2 in malignant transformation and tumorigenesis of Cr(VI)-transformed lung cells.

Medan D, Luanpitpong S, Azad N, Wang L, Jiang BH, Davis ME, Barnett JB, Guo L, Rojanasakul Y.

PLoS One. 2012;7(5):e37045. doi: 10.1371/journal.pone.0037045. Epub 2012 May 29.

PMID:
22666341
[PubMed - indexed for MEDLINE]
Free PMC Article
19.

Arsenic activates EGFR pathway signaling in the lung.

Andrew AS, Mason RA, Memoli V, Duell EJ.

Toxicol Sci. 2009 Jun;109(2):350-7. doi: 10.1093/toxsci/kfp015. Epub 2009 Jan 23.

PMID:
19168569
[PubMed - indexed for MEDLINE]
Free PMC Article
20.

JWA is required for arsenic trioxide induced apoptosis in HeLa and MCF-7 cells via reactive oxygen species and mitochondria linked signal pathway.

Zhou J, Ye J, Zhao X, Li A, Zhou J.

Toxicol Appl Pharmacol. 2008 Jul 1;230(1):33-40. doi: 10.1016/j.taap.2008.01.041. Epub 2008 Feb 20.

PMID:
18387645
[PubMed - indexed for MEDLINE]

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