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Results: 1 to 20 of 320

Similar articles for PubMed (Select 21795680)

1.

Prion protein interacts with BACE1 protein and differentially regulates its activity toward wild type and Swedish mutant amyloid precursor protein.

Griffiths HH, Whitehouse IJ, Baybutt H, Brown D, Kellett KA, Jackson CD, Turner AJ, Piccardo P, Manson JC, Hooper NM.

J Biol Chem. 2011 Sep 23;286(38):33489-500. doi: 10.1074/jbc.M111.278556. Epub 2011 Jul 27.

2.

SNX15 Regulates Cell Surface Recycling of APP and Aβ Generation.

Feng T, Niu M, Ji C, Gao Y, Wen J, Bu G, Xu H, Zhang YW.

Mol Neurobiol. 2015 Jun 27. [Epub ahead of print]

PMID:
26115702
3.

Swedish mutant APP-based BACE1 binding site peptide reduces APP β-cleavage and cerebral Aβ levels in Alzheimer's mice.

Li S, Hou H, Mori T, Sawmiller D, Smith A, Tian J, Wang Y, Giunta B, Sanberg PR, Zhang S, Tan J.

Sci Rep. 2015 Jun 19;5:11322. doi: 10.1038/srep11322.

4.

An Investigation on the Fundamental Interaction between Abeta Peptides and the AT-Rich DNA.

Zhao LN, Zheng J, Chew LY, Mu Y.

J Phys Chem B. 2015 Jul 2;119(26):8247-8259. Epub 2015 Jun 18.

PMID:
26086541
5.

Amyloid-PET predicts inhibition of de novo plaque formation upon chronic γ-secretase modulator treatment.

Brendel M, Jaworska A, Herms J, Trambauer J, Rötzer C, Gildehaus FJ, Carlsen J, Cumming P, Bylund J, Luebbers T, Bartenstein P, Steiner H, Haass C, Baumann K, Rominger A.

Mol Psychiatry. 2015 Jun 9. doi: 10.1038/mp.2015.74. [Epub ahead of print]

PMID:
26055427
6.

The Golgi-Localized γ-Ear-Containing ARF-Binding (GGA) Proteins Alter Amyloid-β Precursor Protein (APP) Processing through Interaction of Their GAE Domain with the Beta-Site APP Cleaving Enzyme 1 (BACE1).

von Einem B, Wahler A, Schips T, Serrano-Pozo A, Proepper C, Boeckers TM, Rueck A, Wirth T, Hyman BT, Danzer KM, Thal DR, von Arnim CA.

PLoS One. 2015 Jun 8;10(6):e0129047. doi: 10.1371/journal.pone.0129047. eCollection 2015.

7.

Differential regulation of the high-affinity choline transporter by wild-type and Swedish mutant amyloid precursor protein.

Cuddy LK, Seah C, Pasternak SH, Rylett RJ.

J Neurochem. 2015 May 13. doi: 10.1111/jnc.13167. [Epub ahead of print]

PMID:
25970623
8.

Clec4g (LSECtin) interacts with BACE1 and suppresses Aβ generation.

Kizuka Y, Kitazume S, Sato K, Taniguchi N.

FEBS Lett. 2015 Jun 4;589(13):1418-22. doi: 10.1016/j.febslet.2015.04.060. Epub 2015 May 7.

PMID:
25957769
9.

Calcium negatively regulates meprin β activity and attenuates substrate cleavage.

Arnold P, Schmidt F, Prox J, Zunke F, Pietrzik C, Lucius R, Becker-Pauly C.

FASEB J. 2015 May 8. pii: fj.15-272310. [Epub ahead of print]

PMID:
25957281
10.

Non-neuronal and neuronal BACE1 elevation in association with angiopathic and leptomeningeal β-amyloid deposition in the human brain.

Xue ZQ, He ZW, Yu JJ, Cai Y, Qiu WY, Pan A, Gai WP, Cai H, Luo XG, Ma C, Yan XX.

BMC Neurol. 2015 May 2;15:71. doi: 10.1186/s12883-015-0327-z.

11.

Endogenous murine Aβ increases amyloid deposition in APP23 but not in APPPS1 transgenic mice.

Mahler J, Morales-Corraliza J, Stolz J, Skodras A, Radde R, Duma CC, Eisele YS, Mazzella MJ, Wong H, Klunk WE, Nilsson KP, Staufenbiel M, Mathews PM, Jucker M, Wegenast-Braun BM.

Neurobiol Aging. 2015 Jul;36(7):2241-7. doi: 10.1016/j.neurobiolaging.2015.03.011. Epub 2015 Mar 25.

PMID:
25911278
12.

The prion protein regulates beta-amyloid-mediated self-renewal of neural stem cells in vitro.

Collins SJ, Tumpach C, Li QX, Lewis V, Ryan TM, Roberts B, Drew SC, Lawson VA, Haigh CL.

Stem Cell Res Ther. 2015 Apr 11;6:60. doi: 10.1186/s13287-015-0067-4.

13.

Structural insight into the mechanism of amyloid precursor protein recognition by β-secretase 1: A molecular dynamics study.

Chakraborty S, Basu S.

Biophys Chem. 2015 Jul;202:1-12. doi: 10.1016/j.bpc.2015.03.006. Epub 2015 Mar 25.

PMID:
25863345
15.

Inhibiting p38 mitogen-activated protein kinase attenuates cerebral ischemic injury in Swedish mutant amyloid precursor protein transgenic mice.

Zou L, Qin H, He Y, Huang H, Lu Y, Chu X.

Neural Regen Res. 2012 May 15;7(14):1088-94. doi: 10.3969/j.issn.1673-5374.2012.14.007.

16.

Aβ reduction in BACE1 heterozygous null 5XFAD mice is associated with transgenic APP level.

Sadleir KR, Eimer WA, Cole SL, Vassar R.

Mol Neurodegener. 2015 Jan 7;10:1. doi: 10.1186/1750-1326-10-1.

17.

The Na+/H+ exchanger NHE6 modulates endosomal pH to control processing of amyloid precursor protein in a cell culture model of Alzheimer disease.

Prasad H, Rao R.

J Biol Chem. 2015 Feb 27;290(9):5311-27. doi: 10.1074/jbc.M114.602219. Epub 2015 Jan 5.

PMID:
25561733
18.

Rapid nuclear transit and impaired degradation of amyloid β and glypican-1-derived heparan sulfate in Tg2576 mouse fibroblasts.

Cheng F, Fransson LÅ, Mani K.

Glycobiology. 2015 May;25(5):548-56. doi: 10.1093/glycob/cwu185. Epub 2014 Dec 19.

PMID:
25527428
19.

SorLA complement-type repeat domains protect the amyloid precursor protein against processing.

Mehmedbasic A, Christensen SK, Nilsson J, Rüetschi U, Gustafsen C, Poulsen AS, Rasmussen RW, Fjorback AN, Larson G, Andersen OM.

J Biol Chem. 2015 Feb 6;290(6):3359-76. doi: 10.1074/jbc.M114.619940. Epub 2014 Dec 18.

PMID:
25525276
20.

Tumor necrosis factor-α reduces beta-amyloid accumulation primarily by lowering cellular prion protein levels in a brain endothelial cell line.

Yasutaka Y, Watanabe T, Nakashima A, Matsumoto J, Futagami K, Yamauchi A, Kataoka Y.

FEBS Lett. 2015 Jan 16;589(2):263-8. doi: 10.1016/j.febslet.2014.12.007. Epub 2014 Dec 10.

PMID:
25497018
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