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Items: 1 to 20 of 170


Prevention of bone marrow cell apoptosis and regulation of hematopoiesis by type I IFNs during systemic responses to pneumocystis lung infection.

Taylor D, Wilkison M, Voyich J, Meissner N.

J Immunol. 2011 May 15;186(10):5956-67. doi: 10.4049/jimmunol.1003558. Epub 2011 Apr 6.


Modulation of inflammasome-mediated pulmonary immune activation by type I IFNs protects bone marrow homeostasis during systemic responses to Pneumocystis lung infection.

Searles S, Gauss K, Wilkison M, Hoyt TR, Dobrinen E, Meissner N.

J Immunol. 2013 Oct 1;191(7):3884-95. doi: 10.4049/jimmunol.1301344. Epub 2013 Aug 23.


Type 1 interferons suppress accelerated osteoclastogenesis and prevent loss of bone mass during systemic inflammatory responses to Pneumocystis lung infection.

Wilkison M, Gauss K, Ran Y, Searles S, Taylor D, Meissner N.

Am J Pathol. 2012 Jul;181(1):151-62. doi: 10.1016/j.ajpath.2012.03.023. Epub 2012 May 22.


Type-I IFN signaling suppresses an excessive IFN-gamma response and thus prevents lung damage and chronic inflammation during Pneumocystis (PC) clearance in CD4 T cell-competent mice.

Meissner N, Swain S, McInnerney K, Han S, Harmsen AG.

Am J Pathol. 2010 Jun;176(6):2806-18. doi: 10.2353/ajpath.2010.091158. Epub 2010 Apr 15.


Role of type I IFNs in pulmonary complications of Pneumocystis murina infection.

Meissner NN, Swain S, Tighe M, Harmsen A, Harmsen A.

J Immunol. 2005 May 1;174(9):5462-71.


Type I interferon signaling and B cells maintain hemopoiesis during Pneumocystis infection of the lung.

Meissner N, Rutkowski M, Harmsen AL, Han S, Harmsen AG.

J Immunol. 2007 May 15;178(10):6604-15.


Type I IFN regulate DC turnover in vivo.

Mattei F, Bracci L, Tough DF, Belardelli F, Schiavoni G.

Eur J Immunol. 2009 Jul;39(7):1807-18. doi: 10.1002/eji.200939233.


Interleukin-2-activated natural killer cells can support hematopoiesis in vitro and promote marrow engraftment in vivo.

Murphy WJ, Keller JR, Harrison CL, Young HA, Longo DL.

Blood. 1992 Aug 1;80(3):670-7.


Granulocyte-macrophage colony-stimulating factor (GM-CSF) induces antiapoptotic and proapoptotic signals in acute myeloid leukemia.

Faderl S, Harris D, Van Q, Kantarjian HM, Talpaz M, Estrov Z.

Blood. 2003 Jul 15;102(2):630-7. Epub 2003 Mar 27.


Granulocyte-macrophage colony-stimulating factor in the innate immune response to Pneumocystis carinii pneumonia in mice.

Paine R 3rd, Preston AM, Wilcoxen S, Jin H, Siu BB, Morris SB, Reed JA, Ross G, Whitsett JA, Beck JM.

J Immunol. 2000 Mar 1;164(5):2602-9.


Bone marrow connexin-43 expression is critical for hematopoietic regeneration after chemotherapy.

Presley CA, Lee AW, Kastl B, Igbinosa I, Yamada Y, Fishman GI, Gutstein DE, Cancelas JA.

Cell Commun Adhes. 2005 Jul-Dec;12(5-6):307-17.


Transforming growth factor-beta1 abrogates Fas-induced growth suppression and apoptosis of murine bone marrow progenitor cells.

Dybedal I, Guan F, Borge OJ, Veiby OP, Ramsfjell V, Nagata S, Jacobsen SE.

Blood. 1997 Nov 1;90(9):3395-403.


In vitro sensitivity of post-bone marrow transplantation CFU-GM and BFU-E to TNF-alpha and IFN-gamma.

Papadakis V, Ferguson KF, Heller G, Kernan NA.

Exp Hematol. 1995 Dec;23(14):1422-30.


TNF receptor signaling contributes to chemokine secretion, inflammation, and respiratory deficits during Pneumocystis pneumonia.

Wright TW, Pryhuber GS, Chess PR, Wang Z, Notter RH, Gigliotti F.

J Immunol. 2004 Feb 15;172(4):2511-21.

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