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Items: 1 to 20 of 110

1.

A virus-binding hot spot on human angiotensin-converting enzyme 2 is critical for binding of two different coronaviruses.

Wu K, Chen L, Peng G, Zhou W, Pennell CA, Mansky LM, Geraghty RJ, Li F.

J Virol. 2011 Jun;85(11):5331-7. doi: 10.1128/JVI.02274-10. Epub 2011 Mar 16.

2.

Identification of residues in the receptor-binding domain (RBD) of the spike protein of human coronavirus NL63 that are critical for the RBD-ACE2 receptor interaction.

Lin HX, Feng Y, Wong G, Wang L, Li B, Zhao X, Li Y, Smaill F, Zhang C.

J Gen Virol. 2008 Apr;89(Pt 4):1015-24. doi: 10.1099/vir.0.83331-0.

PMID:
18343844
3.

Mechanisms of host receptor adaptation by severe acute respiratory syndrome coronavirus.

Wu K, Peng G, Wilken M, Geraghty RJ, Li F.

J Biol Chem. 2012 Mar 16;287(12):8904-11. doi: 10.1074/jbc.M111.325803. Epub 2012 Jan 30.

4.
5.

Human coronavirus EMC does not require the SARS-coronavirus receptor and maintains broad replicative capability in mammalian cell lines.

Müller MA, Raj VS, Muth D, Meyer B, Kallies S, Smits SL, Wollny R, Bestebroer TM, Specht S, Suliman T, Zimmermann K, Binger T, Eckerle I, Tschapka M, Zaki AM, Osterhaus AD, Fouchier RA, Haagmans BL, Drosten C.

MBio. 2012 Dec 11;3(6). pii: e00515-12. doi: 10.1128/mBio.00515-12.

6.

Crystal structure of NL63 respiratory coronavirus receptor-binding domain complexed with its human receptor.

Wu K, Li W, Peng G, Li F.

Proc Natl Acad Sci U S A. 2009 Nov 24;106(47):19970-4. doi: 10.1073/pnas.0908837106. Epub 2009 Nov 9.

7.

Angiotensin-converting enzyme 2 (ACE2) proteins of different bat species confer variable susceptibility to SARS-CoV entry.

Hou Y, Peng C, Yu M, Li Y, Han Z, Li F, Wang LF, Shi Z.

Arch Virol. 2010 Oct;155(10):1563-9. doi: 10.1007/s00705-010-0729-6. Epub 2010 Jun 22.

PMID:
20567988
8.

Interaction of severe acute respiratory syndrome-coronavirus and NL63 coronavirus spike proteins with angiotensin converting enzyme-2.

Mathewson AC, Bishop A, Yao Y, Kemp F, Ren J, Chen H, Xu X, Berkhout B, van der Hoek L, Jones IM.

J Gen Virol. 2008 Nov;89(Pt 11):2741-5. doi: 10.1099/vir.0.2008/003962-0.

9.

Human coronavirus NL63 employs the severe acute respiratory syndrome coronavirus receptor for cellular entry.

Hofmann H, Pyrc K, van der Hoek L, Geier M, Berkhout B, Pöhlmann S.

Proc Natl Acad Sci U S A. 2005 May 31;102(22):7988-93. Epub 2005 May 16.

10.

Effects of human anti-spike protein receptor binding domain antibodies on severe acute respiratory syndrome coronavirus neutralization escape and fitness.

Sui J, Deming M, Rockx B, Liddington RC, Zhu QK, Baric RS, Marasco WA.

J Virol. 2014 Dec;88(23):13769-80. doi: 10.1128/JVI.02232-14. Epub 2014 Sep 17.

11.

Difference in receptor usage between severe acute respiratory syndrome (SARS) coronavirus and SARS-like coronavirus of bat origin.

Ren W, Qu X, Li W, Han Z, Yu M, Zhou P, Zhang SY, Wang LF, Deng H, Shi Z.

J Virol. 2008 Feb;82(4):1899-907. Epub 2007 Dec 12.

12.

Replication-dependent downregulation of cellular angiotensin-converting enzyme 2 protein expression by human coronavirus NL63.

Dijkman R, Jebbink MF, Deijs M, Milewska A, Pyrc K, Buelow E, van der Bijl A, van der Hoek L.

J Gen Virol. 2012 Sep;93(Pt 9):1924-9. doi: 10.1099/vir.0.043919-0. Epub 2012 Jun 20.

PMID:
22718567
13.

SARS coronavirus, but not human coronavirus NL63, utilizes cathepsin L to infect ACE2-expressing cells.

Huang IC, Bosch BJ, Li F, Li W, Lee KH, Ghiran S, Vasilieva N, Dermody TS, Harrison SC, Dormitzer PR, Farzan M, Rottier PJ, Choe H.

J Biol Chem. 2006 Feb 10;281(6):3198-203. Epub 2005 Dec 8.

14.

A hexapeptide of the receptor-binding domain of SARS corona virus spike protein blocks viral entry into host cells via the human receptor ACE2.

Struck AW, Axmann M, Pfefferle S, Drosten C, Meyer B.

Antiviral Res. 2012 Jun;94(3):288-96. doi: 10.1016/j.antiviral.2011.12.012. Epub 2012 Jan 17.

PMID:
22265858
15.

Angiotensin-converting enzyme 2 (ACE2) from raccoon dog can serve as an efficient receptor for the spike protein of severe acute respiratory syndrome coronavirus.

Xu L, Zhang Y, Liu Y, Chen Z, Deng H, Ma Z, Wang H, Hu Z, Deng F.

J Gen Virol. 2009 Nov;90(Pt 11):2695-703. doi: 10.1099/vir.0.013490-0. Epub 2009 Jul 22.

PMID:
19625462
16.

Highly conserved regions within the spike proteins of human coronaviruses 229E and NL63 determine recognition of their respective cellular receptors.

Hofmann H, Simmons G, Rennekamp AJ, Chaipan C, Gramberg T, Heck E, Geier M, Wegele A, Marzi A, Bates P, Pöhlmann S.

J Virol. 2006 Sep;80(17):8639-52.

17.

Endocytosis of the receptor-binding domain of SARS-CoV spike protein together with virus receptor ACE2.

Wang S, Guo F, Liu K, Wang H, Rao S, Yang P, Jiang C.

Virus Res. 2008 Sep;136(1-2):8-15. doi: 10.1016/j.virusres.2008.03.004. Epub 2008 Jun 12.

PMID:
18554741
18.

SARS-CoV replication and pathogenesis in an in vitro model of the human conducting airway epithelium.

Sims AC, Burkett SE, Yount B, Pickles RJ.

Virus Res. 2008 Apr;133(1):33-44. Epub 2007 Apr 23. Review.

19.

Receptor recognition and cross-species infections of SARS coronavirus.

Li F.

Antiviral Res. 2013 Oct;100(1):246-54. doi: 10.1016/j.antiviral.2013.08.014. Epub 2013 Aug 29. Review.

20.

Characterization of the spike protein of human coronavirus NL63 in receptor binding and pseudotype virus entry.

Lin HX, Feng Y, Tu X, Zhao X, Hsieh CH, Griffin L, Junop M, Zhang C.

Virus Res. 2011 Sep;160(1-2):283-93. doi: 10.1016/j.virusres.2011.06.029. Epub 2011 Jul 20.

PMID:
21798295
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