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Items: 1 to 20 of 89

1.

Signaling diversity of PKA achieved via a Ca2+-cAMP-PKA oscillatory circuit.

Ni Q, Ganesan A, Aye-Han NN, Gao X, Allen MD, Levchenko A, Zhang J.

Nat Chem Biol. 2011 Jan;7(1):34-40. doi: 10.1038/nchembio.478. Epub 2010 Nov 21.

2.

Ghrelin attenuates cAMP-PKA signaling to evoke insulinostatic cascade in islet β-cells.

Dezaki K, Damdindorj B, Sone H, Dyachok O, Tengholm A, Gylfe E, Kurashina T, Yoshida M, Kakei M, Yada T.

Diabetes. 2011 Sep;60(9):2315-24. doi: 10.2337/db11-0368. Epub 2011 Jul 25.

3.

cAMP oscillations restrict protein kinase A redistribution in insulin-secreting cells.

Dyachok O, Sågetorp J, Isakov Y, Tengholm A.

Biochem Soc Trans. 2006 Aug;34(Pt 4):498-501.

PMID:
16856843
4.

cAMP mediators of pulsatile insulin secretion from glucose-stimulated single beta-cells.

Idevall-Hagren O, Barg S, Gylfe E, Tengholm A.

J Biol Chem. 2010 Jul 23;285(30):23007-18. doi: 10.1074/jbc.M109.095992. Epub 2010 May 24.

5.

Adenylate cyclase/cAMP/protein kinase A signaling pathway inhibits endothelin type A receptor-operated Ca²⁺ entry mediated via transient receptor potential canonical 6 channels.

Horinouchi T, Higa T, Aoyagi H, Nishiya T, Terada K, Miwa S.

J Pharmacol Exp Ther. 2012 Jan;340(1):143-51. doi: 10.1124/jpet.111.187500. Epub 2011 Oct 14.

6.

Coupling of cAMP/PKA and MAPK signaling in neuronal cells is dependent on developmental stage.

Vogt Weisenhorn DM, Roback LJ, Kwon JH, Wainer BH.

Exp Neurol. 2001 May;169(1):44-55.

PMID:
11312557
7.

Phosphatidylinositol 3-kinase offsets cAMP-mediated positive inotropic effect via inhibiting Ca2+ influx in cardiomyocytes.

Leblais V, Jo SH, Chakir K, Maltsev V, Zheng M, Crow MT, Wang W, Lakatta EG, Xiao RP.

Circ Res. 2004 Dec 10;95(12):1183-90. Epub 2004 Nov 11.

8.

cAMP-mediated signaling normalizes glucose-stimulated insulin secretion in uncoupling protein-2 overexpressing beta-cells.

McQuaid TS, Saleh MC, Joseph JW, Gyulkhandanyan A, Manning-Fox JE, MacLellan JD, Wheeler MB, Chan CB.

J Endocrinol. 2006 Sep;190(3):669-80.

9.

Low, but physiological, concentration of GLP-1 stimulates insulin secretion independent of the cAMP-dependent protein kinase pathway.

Shigeto M, Katsura M, Matsuda M, Ohkuma S, Kaku K.

J Pharmacol Sci. 2008 Nov;108(3):274-9. Epub 2008 Nov 6.

10.

β-Cell-specific protein kinase A activation enhances the efficiency of glucose control by increasing acute-phase insulin secretion.

Kaihara KA, Dickson LM, Jacobson DA, Tamarina N, Roe MW, Philipson LH, Wicksteed B.

Diabetes. 2013 May;62(5):1527-36. doi: 10.2337/db12-1013. Epub 2013 Jan 24.

12.
13.

Oscillations of cyclic AMP in hormone-stimulated insulin-secreting beta-cells.

Dyachok O, Isakov Y, Sågetorp J, Tengholm A.

Nature. 2006 Jan 19;439(7074):349-52.

PMID:
16421574
14.

Stimulation of Ca2+ influx in alpha T3-1 gonadotrophs via the cAMP/PKA signaling system.

Hezareh M, Schlegel W, Rawlings SR.

Am J Physiol. 1997 Nov;273(5 Pt 1):E850-8.

15.
16.

Protein kinase A-dependent and -independent signaling pathways contribute to cyclic AMP-stimulated proliferation.

Cass LA, Summers SA, Prendergast GV, Backer JM, Birnbaum MJ, Meinkoth JL.

Mol Cell Biol. 1999 Sep;19(9):5882-91.

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20.

A key phosphorylation site in AC8 mediates regulation of Ca(2+)-dependent cAMP dynamics by an AC8-AKAP79-PKA signalling complex.

Willoughby D, Halls ML, Everett KL, Ciruela A, Skroblin P, Klussmann E, Cooper DM.

J Cell Sci. 2012 Dec 1;125(Pt 23):5850-9. doi: 10.1242/jcs.111427. Epub 2012 Sep 12.

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