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Similar articles for PubMed (Select 20702563)


The role of Drosophila Lamin C in muscle function and gene expression.

Dialynas G, Speese S, Budnik V, Geyer PK, Wallrath LL.

Development. 2010 Sep;137(18):3067-77. doi: 10.1242/dev.048231. Epub 2010 Aug 11.


A comparative study of Drosophila and human A-type lamins.

Schulze SR, Curio-Penny B, Speese S, Dialynas G, Cryderman DE, McDonough CW, Nalbant D, Petersen M, Budnik V, Geyer PK, Wallrath LL.

PLoS One. 2009 Oct 26;4(10):e7564. doi: 10.1371/journal.pone.0007564.


Molecular genetic analysis of the nested Drosophila melanogaster lamin C gene.

Schulze SR, Curio-Penny B, Li Y, Imani RA, Rydberg L, Geyer PK, Wallrath LL.

Genetics. 2005 Sep;171(1):185-96. Epub 2005 Jun 18.


Expression and localization of nuclear proteins in autosomal-dominant Emery-Dreifuss muscular dystrophy with LMNA R377H mutation.

Reichart B, Klafke R, Dreger C, Krüger E, Motsch I, Ewald A, Schäfer J, Reichmann H, Müller CR, Dabauvalle MC.

BMC Cell Biol. 2004 Mar 30;5:12.


LMNA variants cause cytoplasmic distribution of nuclear pore proteins in Drosophila and human muscle.

Dialynas G, Flannery KM, Zirbel LN, Nagy PL, Mathews KD, Moore SA, Wallrath LL.

Hum Mol Genet. 2012 Apr 1;21(7):1544-56. doi: 10.1093/hmg/ddr592. Epub 2011 Dec 20.


Loss of Drosophila A-type lamin C initially causes tendon abnormality including disintegration of cytoskeleton and nuclear lamina in muscular defects.

Uchino R, Nonaka YK, Horigome T, Sugiyama S, Furukawa K.

Dev Biol. 2013 Jan 1;373(1):216-27. doi: 10.1016/j.ydbio.2012.08.001. Epub 2012 Sep 13.


Expression of lamin A mutated in the carboxyl-terminal tail generates an aberrant nuclear phenotype similar to that observed in cells from patients with Dunnigan-type partial lipodystrophy and Emery-Dreifuss muscular dystrophy.

Favreau C, Dubosclard E, Ostlund C, Vigouroux C, Capeau J, Wehnert M, Higuet D, Worman HJ, Courvalin JC, Buendia B.

Exp Cell Res. 2003 Jan 1;282(1):14-23.


Nuclear titin interacts with A- and B-type lamins in vitro and in vivo.

Zastrow MS, Flaherty DB, Benian GM, Wilson KL.

J Cell Sci. 2006 Jan 15;119(Pt 2):239-49.


Myopathic lamin mutations impair nuclear stability in cells and tissue and disrupt nucleo-cytoskeletal coupling.

Zwerger M, Jaalouk DE, Lombardi ML, Isermann P, Mauermann M, Dialynas G, Herrmann H, Wallrath LL, Lammerding J.

Hum Mol Genet. 2013 Jun 15;22(12):2335-49. doi: 10.1093/hmg/ddt079. Epub 2013 Feb 19.


Expression of Drosophila lamin C is developmentally regulated: analogies with vertebrate A-type lamins.

Riemer D, Stuurman N, Berrios M, Hunter C, Fisher PA, Weber K.

J Cell Sci. 1995 Oct;108 ( Pt 10):3189-98.


The lamin CxxM motif promotes nuclear membrane growth.

Prüfert K, Vogel A, Krohne G.

J Cell Sci. 2004 Dec 1;117(Pt 25):6105-16. Epub 2004 Nov 16.


Effects of expressing lamin A mutant protein causing Emery-Dreifuss muscular dystrophy and familial partial lipodystrophy in HeLa cells.

Bechert K, Lagos-Quintana M, Harborth J, Weber K, Osborn M.

Exp Cell Res. 2003 May 15;286(1):75-86.


Characterization of lamin mutation phenotypes in Drosophila and comparison to human laminopathies.

Muñoz-Alarcón A, Pavlovic M, Wismar J, Schmitt B, Eriksson M, Kylsten P, Dushay MS.

PLoS One. 2007 Jun 13;2(6):e532.


Lamin A N-terminal phosphorylation is associated with myoblast activation: impairment in Emery-Dreifuss muscular dystrophy.

Cenni V, Sabatelli P, Mattioli E, Marmiroli S, Capanni C, Ognibene A, Squarzoni S, Maraldi NM, Bonne G, Columbaro M, Merlini L, Lattanzi G.

J Med Genet. 2005 Mar;42(3):214-20.


The nuclear membrane and mechanotransduction: impaired nuclear mechanics and mechanotransduction in lamin A/C deficient cells.

Lammerding J, Lee RT.

Novartis Found Symp. 2005;264:264-73; discussion 273-8. Review.


Both lamin A and lamin C mutations cause lamina instability as well as loss of internal nuclear lamin organization.

Broers JL, Kuijpers HJ, Ostlund C, Worman HJ, Endert J, Ramaekers FC.

Exp Cell Res. 2005 Apr 1;304(2):582-92. Epub 2004 Dec 20.


Increased solubility of lamins and redistribution of lamin C in X-linked Emery-Dreifuss muscular dystrophy fibroblasts.

Markiewicz E, Venables R, Mauricio-Alvarez-Reyes, Quinlan R, Dorobek M, Hausmanowa-Petrucewicz I, Hutchison C.

J Struct Biol. 2002 Oct-Dec;140(1-3):241-53.


Lamins A and C are differentially dysfunctional in autosomal dominant Emery-Dreifuss muscular dystrophy.

Motsch I, Kaluarachchi M, Emerson LJ, Brown CA, Brown SC, Dabauvalle MC, Ellis JA.

Eur J Cell Biol. 2005 Sep;84(9):765-81.


The Ig-like structure of the C-terminal domain of lamin A/C, mutated in muscular dystrophies, cardiomyopathy, and partial lipodystrophy.

Krimm I, Ostlund C, Gilquin B, Couprie J, Hossenlopp P, Mornon JP, Bonne G, Courvalin JC, Worman HJ, Zinn-Justin S.

Structure. 2002 Jun;10(6):811-23.

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