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Results: 1 to 20 of 106

Related Citations for PubMed (Select 20637282)

1.

Aspartoacylase deficiency affects early postnatal development of oligodendrocytes and myelination.

Mattan NS, Ghiani CA, Lloyd M, Matalon R, Bok D, Casaccia P, de Vellis J.

Neurobiol Dis. 2010 Nov;40(2):432-43. doi: 10.1016/j.nbd.2010.07.003. Epub 2010 Jul 14.

2.

Nur7 is a nonsense mutation in the mouse aspartoacylase gene that causes spongy degeneration of the CNS.

Traka M, Wollmann RL, Cerda SR, Dugas J, Barres BA, Popko B.

J Neurosci. 2008 Nov 5;28(45):11537-49. doi: 10.1523/JNEUROSCI.1490-08.2008.

3.

Lack of aspartoacylase activity disrupts survival and differentiation of neural progenitors and oligodendrocytes in a mouse model of Canavan disease.

Kumar S, Biancotti JC, Matalon R, de Vellis J.

J Neurosci Res. 2009 Nov 15;87(15):3415-27. doi: 10.1002/jnr.22233.

PMID:
19739253
4.

Canavan disease: a white matter disorder.

Kumar S, Mattan NS, de Vellis J.

Ment Retard Dev Disabil Res Rev. 2006;12(2):157-65. Review.

PMID:
16807907
5.

Restoration of aspartoacylase activity in CNS neurons does not ameliorate motor deficits and demyelination in a model of Canavan disease.

Klugmann M, Leichtlein CB, Symes CW, Serikawa T, Young D, During MJ.

Mol Ther. 2005 May;11(5):745-53.

PMID:
15851013
6.

Defective N-acetylaspartate catabolism reduces brain acetate levels and myelin lipid synthesis in Canavan's disease.

Madhavarao CN, Arun P, Moffett JR, Szucs S, Surendran S, Matalon R, Garbern J, Hristova D, Johnson A, Jiang W, Namboodiri MA.

Proc Natl Acad Sci U S A. 2005 Apr 5;102(14):5221-6. Epub 2005 Mar 22.

7.

Peroxisome-proliferator-activated receptor gamma coactivator 1 α contributes to dysmyelination in experimental models of Huntington's disease.

Xiang Z, Valenza M, Cui L, Leoni V, Jeong HK, Brilli E, Zhang J, Peng Q, Duan W, Reeves SA, Cattaneo E, Krainc D.

J Neurosci. 2011 Jun 29;31(26):9544-53. doi: 10.1523/JNEUROSCI.1291-11.2011.

8.

Loss of central auditory processing in a mouse model of Canavan disease.

von Jonquieres G, Froud KE, Klugmann CB, Wong AC, Housley GD, Klugmann M.

PLoS One. 2014 May 14;9(5):e97374. doi: 10.1371/journal.pone.0097374. eCollection 2014.

9.

Immunohistochemical localization of aspartoacylase in the rat central nervous system.

Madhavarao CN, Moffett JR, Moore RA, Viola RE, Namboodiri MA, Jacobowitz DM.

J Comp Neurol. 2004 May 3;472(3):318-29.

PMID:
15065127
10.

Adeno-associated virus-mediated aspartoacylase gene transfer to the brain of knockout mouse for canavan disease.

Matalon R, Surendran S, Rady PL, Quast MJ, Campbell GA, Matalon KM, Tyring SK, Wei J, Peden CS, Ezell EL, Muzyczka N, Mandel RJ.

Mol Ther. 2003 May;7(5 Pt 1):580-7.

PMID:
12718900
11.

Galactolipid deficiency in the early pathogenesis of neuronal ceroid lipofuscinosis model Cln8mnd : implications to delayed myelination and oligodendrocyte maturation.

Kuronen M, Hermansson M, Manninen O, Zech I, Talvitie M, Laitinen T, Gröhn O, Somerharju P, Eckhardt M, Cooper JD, Lehesjoki AE, Lahtinen U, Kopra O.

Neuropathol Appl Neurobiol. 2012 Aug;38(5):471-86. doi: 10.1111/j.1365-2990.2011.01233.x.

PMID:
22044361
12.

Developmental increase of aspartoacylase in oligodendrocytes parallels CNS myelination.

Kirmani BF, Jacobowitz DM, Namboodiri MA.

Brain Res Dev Brain Res. 2003 Jan 10;140(1):105-15.

PMID:
12524181
13.

Glial promoter selectivity following AAV-delivery to the immature brain.

von Jonquieres G, Mersmann N, Klugmann CB, Harasta AE, Lutz B, Teahan O, Housley GD, Fröhlich D, Krämer-Albers EM, Klugmann M.

PLoS One. 2013 Jun 14;8(6):e65646. doi: 10.1371/journal.pone.0065646. Print 2013.

14.

Dysmyelination and reduced myelin basic protein gene expression by oligodendrocytes of SHP-1-deficient mice.

Massa PT, Wu C, Fecenko-Tacka K.

J Neurosci Res. 2004 Jul 1;77(1):15-25.

PMID:
15197735
15.

Aspartoacylase gene knockout results in severe vacuolation in the white matter and gray matter of the spinal cord in the mouse.

Surendran S, Campbell GA, Tyring SK, Matalon R.

Neurobiol Dis. 2005 Mar;18(2):385-9.

PMID:
15686967
16.

Canavan disease: a monogenic trait with complex genomic interaction.

Surendran S, Michals-Matalon K, Quast MJ, Tyring SK, Wei J, Ezell EL, Matalon R.

Mol Genet Metab. 2003 Sep-Oct;80(1-2):74-80. Review. Erratum in: Mol Genet Metab. 2006 Mar;87(3):279.

PMID:
14567959
17.

Canavan disease: studies on the knockout mouse.

Matalon R, Michals-Matalon K, Surendran S, Tyring SK.

Adv Exp Med Biol. 2006;576:77-93; discussion 361-3. Review.

PMID:
16802706
18.

Dietary triheptanoin rescues oligodendrocyte loss, dysmyelination and motor function in the nur7 mouse model of Canavan disease.

Francis JS, Markov V, Leone P.

J Inherit Metab Dis. 2014 May;37(3):369-81. doi: 10.1007/s10545-013-9663-6. Epub 2013 Nov 28.

PMID:
24288037
19.

Aspartoacylase gene knockout in the mouse: impact on reproduction.

Surendran S, Szucs S, Tyring SK, Matalon R.

Reprod Toxicol. 2005 Jul-Aug;20(2):281-3.

PMID:
15907664
20.

Are astrocytes the missing link between lack of brain aspartoacylase activity and the spongiform leukodystrophy in Canavan disease?

Baslow MH, Guilfoyle DN.

Neurochem Res. 2009 Sep;34(9):1523-34. doi: 10.1007/s11064-009-9958-z. Epub 2009 Mar 25.

PMID:
19319678
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