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Items: 1 to 20 of 681

1.

Survival motor neuron deficiency enhances progression in an amyotrophic lateral sclerosis mouse model.

Turner BJ, Parkinson NJ, Davies KE, Talbot K.

Neurobiol Dis. 2009 Jun;34(3):511-7. doi: 10.1016/j.nbd.2009.03.005. Epub 2009 Mar 28.

PMID:
19332122
2.

Overexpression of survival motor neuron improves neuromuscular function and motor neuron survival in mutant SOD1 mice.

Turner BJ, Alfazema N, Sheean RK, Sleigh JN, Davies KE, Horne MK, Talbot K.

Neurobiol Aging. 2014 Apr;35(4):906-15. doi: 10.1016/j.neurobiolaging.2013.09.030. Epub 2013 Oct 24.

3.

Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies.

Kariya S, Re DB, Jacquier A, Nelson K, Przedborski S, Monani UR.

Hum Mol Genet. 2012 Aug 1;21(15):3421-34. doi: 10.1093/hmg/dds174. Epub 2012 May 11.

4.

TDP-43 expression in mouse models of amyotrophic lateral sclerosis and spinal muscular atrophy.

Turner BJ, Bäumer D, Parkinson NJ, Scaber J, Ansorge O, Talbot K.

BMC Neurosci. 2008 Oct 28;9:104. doi: 10.1186/1471-2202-9-104.

5.
6.

Dismutase-competent SOD1 mutant accumulation in myelinating Schwann cells is not detrimental to normal or transgenic ALS model mice.

Turner BJ, Ackerley S, Davies KE, Talbot K.

Hum Mol Genet. 2010 Mar 1;19(5):815-24. doi: 10.1093/hmg/ddp550. Epub 2009 Dec 15.

7.

Knocking down metabotropic glutamate receptor 1 improves survival and disease progression in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Milanese M, Giribaldi F, Melone M, Bonifacino T, Musante I, Carminati E, Rossi PI, Vergani L, Voci A, Conti F, Puliti A, Bonanno G.

Neurobiol Dis. 2014 Apr;64:48-59. doi: 10.1016/j.nbd.2013.11.006. Epub 2013 Dec 19.

8.

Differential effects of mutant SOD1 on protein structure of skeletal muscle and spinal cord of familial amyotrophic lateral sclerosis: role of chaperone network.

Wei R, Bhattacharya A, Hamilton RT, Jernigan AL, Chaudhuri AR.

Biochem Biophys Res Commun. 2013 Aug 16;438(1):218-23. doi: 10.1016/j.bbrc.2013.07.060. Epub 2013 Jul 23.

PMID:
23886956
9.

Over-expression of Hsp27 does not influence disease in the mutant SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Krishnan J, Vannuvel K, Andries M, Waelkens E, Robberecht W, Van Den Bosch L.

J Neurochem. 2008 Sep;106(5):2170-83. doi: 10.1111/j.1471-4159.2008.05545.x. Epub 2008 Jul 4.

11.

Microglia and motor neurons during disease progression in the SOD1G93A mouse model of amyotrophic lateral sclerosis: changes in arginase1 and inducible nitric oxide synthase.

Lewis KE, Rasmussen AL, Bennett W, King A, West AK, Chung RS, Chuah MI.

J Neuroinflammation. 2014 Mar 23;11:55. doi: 10.1186/1742-2094-11-55.

12.

Functional alterations of the ubiquitin-proteasome system in motor neurons of a mouse model of familial amyotrophic lateral sclerosis.

Cheroni C, Marino M, Tortarolo M, Veglianese P, De Biasi S, Fontana E, Zuccarello LV, Maynard CJ, Dantuma NP, Bendotti C.

Hum Mol Genet. 2009 Jan 1;18(1):82-96. doi: 10.1093/hmg/ddn319. Epub 2008 Sep 29.

13.

SMN genotypes producing less SMN protein increase susceptibility to and severity of sporadic ALS.

Veldink JH, Kalmijn S, Van der Hout AH, Lemmink HH, Groeneveld GJ, Lummen C, Scheffer H, Wokke JH, Van den Berg LH.

Neurology. 2005 Sep 27;65(6):820-5. Epub 2005 Aug 10.

PMID:
16093455
14.

Transcriptional profiling in the lumbar spinal cord of a mouse model of amyotrophic lateral sclerosis: a role for wild-type superoxide dismutase 1 in sporadic disease?

D'Arrigo A, Colavito D, Peña-Altamira E, Fabris M, Dam M, Contestabile A, Leon A.

J Mol Neurosci. 2010 Jul;41(3):404-15. doi: 10.1007/s12031-010-9332-2. Epub 2010 Feb 23. Erratum in: J Mol Neurosci. 2010 Jul;41(3):416.

PMID:
20177826
15.
16.

Redox system expression in the motor neurons in amyotrophic lateral sclerosis (ALS): immunohistochemical studies on sporadic ALS, superoxide dismutase 1 (SOD1)-mutated familial ALS, and SOD1-mutated ALS animal models.

Kato S, Kato M, Abe Y, Matsumura T, Nishino T, Aoki M, Itoyama Y, Asayama K, Awaya A, Hirano A, Ohama E.

Acta Neuropathol. 2005 Aug;110(2):101-12. Epub 2005 Jun 28.

PMID:
15983830
17.

Overexpression of Abeta is associated with acceleration of onset of motor impairment and superoxide dismutase 1 aggregation in an amyotrophic lateral sclerosis mouse model.

Li QX, Mok SS, Laughton KM, McLean CA, Volitakis I, Cherny RA, Cheung NS, White AR, Masters CL.

Aging Cell. 2006 Apr;5(2):153-65.

18.

Molecular chaperone mediated late-stage neuroprotection in the SOD1(G93A) mouse model of amyotrophic lateral sclerosis.

Novoselov SS, Mustill WJ, Gray AL, Dick JR, Kanuga N, Kalmar B, Greensmith L, Cheetham ME.

PLoS One. 2013 Aug 30;8(8):e73944. doi: 10.1371/journal.pone.0073944. eCollection 2013.

19.

In vivo quantification of spinal and bulbar motor neuron degeneration in the G93A-SOD1 transgenic mouse model of ALS by T2 relaxation time and apparent diffusion coefficient.

Niessen HG, Angenstein F, Sander K, Kunz WS, Teuchert M, Ludolph AC, Heinze HJ, Scheich H, Vielhaber S.

Exp Neurol. 2006 Oct;201(2):293-300. Epub 2006 Jun 5.

PMID:
16740261
20.

H63D HFE genotype accelerates disease progression in animal models of amyotrophic lateral sclerosis.

Nandar W, Neely EB, Simmons Z, Connor JR.

Biochim Biophys Acta. 2014 Dec;1842(12 Pt A):2413-26. doi: 10.1016/j.bbadis.2014.09.016. Epub 2014 Oct 5.

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