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Items: 1 to 20 of 1020

1.

Inflammation markers are modulated by responses to diets differing in postprandial insulin responses in individuals with the metabolic syndrome.

Kallio P, Kolehmainen M, Laaksonen DE, Pulkkinen L, Atalay M, Mykkänen H, Uusitupa M, Poutanen K, Niskanen L.

Am J Clin Nutr. 2008 May;87(5):1497-503.

2.

Dietary carbohydrate modification induces alterations in gene expression in abdominal subcutaneous adipose tissue in persons with the metabolic syndrome: the FUNGENUT Study.

Kallio P, Kolehmainen M, Laaksonen DE, Kekäläinen J, Salopuro T, Sivenius K, Pulkkinen L, Mykkänen HM, Niskanen L, Uusitupa M, Poutanen KS.

Am J Clin Nutr. 2007 May;85(5):1417-27.

3.

Dietary carbohydrate modification enhances insulin secretion in persons with the metabolic syndrome.

Laaksonen DE, Toppinen LK, Juntunen KS, Autio K, Liukkonen KH, Poutanen KS, Niskanen L, Mykkänen HM.

Am J Clin Nutr. 2005 Dec;82(6):1218-27.

4.

Postprandial glucose, insulin, and incretin responses to grain products in healthy subjects.

Juntunen KS, Niskanen LK, Liukkonen KH, Poutanen KS, Holst JJ, Mykkänen HM.

Am J Clin Nutr. 2002 Feb;75(2):254-62.

5.

Effects of breakfast meal composition on second meal metabolic responses in adults with Type 2 diabetes mellitus.

Clark CA, Gardiner J, McBurney MI, Anderson S, Weatherspoon LJ, Henry DN, Hord NG.

Eur J Clin Nutr. 2006 Sep;60(9):1122-9. Epub 2006 May 3.

PMID:
16670695
6.

Factors related to colonic fermentation of nondigestible carbohydrates of a previous evening meal increase tissue glucose uptake and moderate glucose-associated inflammation.

Priebe MG, Wang H, Weening D, Schepers M, Preston T, Vonk RJ.

Am J Clin Nutr. 2010 Jan;91(1):90-7. doi: 10.3945/ajcn.2009.28521. Epub 2009 Nov 4.

7.

Interaction between cholesterol and glucose metabolism during dietary carbohydrate modification in subjects with the metabolic syndrome.

Hallikainen M, Toppinen L, Mykkänen H, Agren JJ, Laaksonen DE, Miettinen TA, Niskanen L, Poutanen KS, Gylling H.

Am J Clin Nutr. 2006 Dec;84(6):1385-92.

8.

Vinegar supplementation lowers glucose and insulin responses and increases satiety after a bread meal in healthy subjects.

Ostman E, Granfeldt Y, Persson L, Björck I.

Eur J Clin Nutr. 2005 Sep;59(9):983-8.

PMID:
16015276
9.

Rye bread decreases postprandial insulin response but does not alter glucose response in healthy Finnish subjects.

Leinonen K, Liukkonen K, Poutanen K, Uusitupa M, Mykkänen H.

Eur J Clin Nutr. 1999 Apr;53(4):262-7.

PMID:
10334650
11.

Rye products in the diabetic diet. Postprandial glucose and hormonal responses in non-insulin-dependent diabetic patients as compared to starch availability in vitro and experiments in rats.

Hagander B, Björck I, Asp NG, Efendić S, Holm J, Nilsson-Ehle P, Lundquist I, Scherstén B.

Diabetes Res Clin Pract. 1987 Mar-Apr;3(2):85-96.

PMID:
2436870
12.

Oat bran concentrate bread products improve long-term control of diabetes: a pilot study.

Pick ME, Hawrysh ZJ, Gee MI, Toth E, Garg ML, Hardin RT.

J Am Diet Assoc. 1996 Dec;96(12):1254-61.

PMID:
8948386
13.

Whole-grain cereal products based on a high-fibre barley or oat genotype lower post-prandial glucose and insulin responses in healthy humans.

Alminger M, Eklund-Jonsson C.

Eur J Nutr. 2008 Sep;47(6):294-300. doi: 10.1007/s00394-008-0724-9. Epub 2008 Jul 16.

PMID:
18633670
14.

Glycemic responses of oat bran products in type 2 diabetic patients.

Tapola N, Karvonen H, Niskanen L, Mikola M, Sarkkinen E.

Nutr Metab Cardiovasc Dis. 2005 Aug;15(4):255-61.

PMID:
16054549
15.

The acute impact of ingestion of breads of varying composition on blood glucose, insulin and incretins following first and second meals.

Najjar AM, Parsons PM, Duncan AM, Robinson LE, Yada RY, Graham TE.

Br J Nutr. 2009 Feb;101(3):391-8. doi: 10.1017/S0007114508003085. Epub 2008 Jun 23.

PMID:
18570696
16.

Postprandial glucose metabolism and SCFA after consuming wholegrain rye bread and wheat bread enriched with bioprocessed rye bran in individuals with mild gastrointestinal symptoms.

Lappi J, Mykkänen H, Bach Knudsen KE, Kirjavainen P, Katina K, Pihlajamäki J, Poutanen K, Kolehmainen M.

Nutr J. 2014 Nov 4;13:104. doi: 10.1186/1475-2891-13-104.

17.

Glycemic and insulinemic meal responses modulate postprandial hepatic and intestinal lipoprotein accumulation in obese, insulin-resistant subjects.

Harbis A, Perdreau S, Vincent-Baudry S, Charbonnier M, Bernard MC, Raccah D, Senft M, Lorec AM, Defoort C, Portugal H, Vinoy S, Lang V, Lairon D.

Am J Clin Nutr. 2004 Oct;80(4):896-902.

18.

Characteristics of some wheat-based foods of the Italian diet in relation to their influence on postprandial glucose metabolism in patients with type 2 diabetes.

Giacco R, Brighenti F, Parillo M, Capuano M, Ciardullo AV, Rivieccio A, Rivellese AA, Riccardi G.

Br J Nutr. 2001 Jan;85(1):33-40.

PMID:
11227031
19.

Plasma ghrelin levels after two high-carbohydrate meals producing different insulin responses in patients with metabolic syndrome.

Heinonen MV, Karhunen LJ, Chabot ED, Toppinen LK, Juntunen KS, Laaksonen DE, Siloaho M, Liukkonen KH, Herzig KH, Niskanen LK, Mykkänen HM.

Regul Pept. 2007 Feb 1;138(2-3):118-25. Epub 2006 Oct 9.

PMID:
17030069
20.

Structural differences between rye and wheat breads but not total fiber content may explain the lower postprandial insulin response to rye bread.

Juntunen KS, Laaksonen DE, Autio K, Niskanen LK, Holst JJ, Savolainen KE, Liukkonen KH, Poutanen KS, Mykkänen HM.

Am J Clin Nutr. 2003 Nov;78(5):957-64.

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