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Items: 1 to 20 of 99

1.

Genome-wide analysis of cellular response to bacterial genotoxin CdtB in yeast.

Kitagawa T, Hoshida H, Akada R.

Infect Immun. 2007 Mar;75(3):1393-402. Epub 2007 Jan 12.

2.

Cytolethal distending toxin from Aggregatibacter actinomycetemcomitans induces DNA damage, S/G2 cell cycle arrest, and caspase- independent death in a Saccharomyces cerevisiae model.

Matangkasombut O, Wattanawaraporn R, Tsuruda K, Ohara M, Sugai M, Mongkolsuk S.

Infect Immun. 2010 Feb;78(2):783-92. doi: 10.1128/IAI.00857-09. Epub 2009 Dec 7.

4.

Comparative genome-wide screening identifies a conserved doxorubicin repair network that is diploid specific in Saccharomyces cerevisiae.

Westmoreland TJ, Wickramasekara SM, Guo AY, Selim AL, Winsor TS, Greenleaf AL, Blackwell KL, Olson JA Jr, Marks JR, Bennett CB.

PLoS One. 2009 Jun 8;4(6):e5830. doi: 10.1371/journal.pone.0005830.

5.

Cytolethal distending toxin demonstrates genotoxic activity in a yeast model.

Hassane DC, Lee RB, Mendenhall MD, Pickett CL.

Infect Immun. 2001 Sep;69(9):5752-9.

6.

Cytolethal distending toxin: a conserved bacterial genotoxin that blocks cell cycle progression, leading to apoptosis of a broad range of mammalian cell lineages.

Jinadasa RN, Bloom SE, Weiss RS, Duhamel GE.

Microbiology. 2011 Jul;157(Pt 7):1851-75. doi: 10.1099/mic.0.049536-0. Epub 2011 May 12. Review.

7.

The contribution of cytolethal distending toxin to bacterial pathogenesis.

Smith JL, Bayles DO.

Crit Rev Microbiol. 2006 Oct-Dec;32(4):227-48. Review.

PMID:
17123907
8.

The DNA-damage signature in Saccharomyces cerevisiae is associated with single-strand breaks in DNA.

Fry RC, DeMott MS, Cosgrove JP, Begley TJ, Samson LD, Dedon PC.

BMC Genomics. 2006 Dec 12;7:313.

9.

Bacterial genotoxin triggers FEN1-dependent RhoA activation, cytoskeleton remodeling and cell survival.

Guerra L, Guidi R, Slot I, Callegari S, Sompallae R, Pickett CL, Åström S, Eisele F, Wolf D, Sjögren C, Masucci MG, Frisan T.

J Cell Sci. 2011 Aug 15;124(Pt 16):2735-42. doi: 10.1242/jcs.085845.

10.

The Saccharomyces cerevisiae Ku autoantigen homologue affects radiosensitivity only in the absence of homologous recombination.

Siede W, Friedl AA, Dianova I, Eckardt-Schupp F, Friedberg EC.

Genetics. 1996 Jan;142(1):91-102.

11.

SIR functions are required for the toleration of an unrepaired double-strand break in a dispensable yeast chromosome.

Bennett CB, Snipe JR, Westmoreland JW, Resnick MA.

Mol Cell Biol. 2001 Aug;21(16):5359-73.

13.

Bacterial toxin modulation of the eukaryotic cell cycle: are all cytolethal distending toxins created equally?

Gargi A, Reno M, Blanke SR.

Front Cell Infect Microbiol. 2012 Oct 8;2:124. doi: 10.3389/fcimb.2012.00124. eCollection 2012. Review.

14.

Detection and functionality of the CdtB, PltA, and PltB from Salmonella enterica serovar Javiana.

Mezal EH, Bae D, Khan AA.

Pathog Dis. 2014 Nov;72(2):95-103. doi: 10.1111/2049-632X.12191. Epub 2014 Jul 16.

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Switching yeast from meiosis to mitosis: double-strand break repair, recombination and synaptonemal complex.

Zenvirth D, Loidl J, Klein S, Arbel A, Shemesh R, Simchen G.

Genes Cells. 1997 Aug;2(8):487-98.

19.

An N-terminal segment of the active component of the bacterial genotoxin cytolethal distending toxin B (CDTB) directs CDTB into the nucleus.

Nishikubo S, Ohara M, Ueno Y, Ikura M, Kurihara H, Komatsuzawa H, Oswald E, Sugai M.

J Biol Chem. 2003 Dec 12;278(50):50671-81. Epub 2003 Aug 28.

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