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Results: 1 to 20 of 161

Similar articles for PubMed (Select 16479004)

1.

Mammalian Rad9 plays a role in telomere stability, S- and G2-phase-specific cell survival, and homologous recombinational repair.

Pandita RK, Sharma GG, Laszlo A, Hopkins KM, Davey S, Chakhparonian M, Gupta A, Wellinger RJ, Zhang J, Powell SN, Roti Roti JL, Lieberman HB, Pandita TK.

Mol Cell Biol. 2006 Mar;26(5):1850-64.

2.

Role of mammalian Rad9 in genomic stability and ionizing radiation response.

Pandita TK.

Cell Cycle. 2006 Jun;5(12):1289-91. Epub 2006 Jun 15.

PMID:
16760671
4.
5.

Cell cycle-dependent processing of DNA lesions controls localization of Rad9 to sites of genotoxic stress.

Warmerdam DO, Freire R, Kanaar R, Smits VA.

Cell Cycle. 2009 Jun 1;8(11):1765-74. Epub 2009 Jun 10.

PMID:
19411845
6.
7.

Alteration/deficiency in activation-3 (Ada3) plays a critical role in maintaining genomic stability.

Mirza S, Katafiasz BJ, Kumar R, Wang J, Mohibi S, Jain S, Gurumurthy CB, Pandita TK, Dave BJ, Band H, Band V.

Cell Cycle. 2012 Nov 15;11(22):4266-74. doi: 10.4161/cc.22613. Epub 2012 Oct 24.

8.

Nuclear factories for signalling and repairing DNA double strand breaks in living fission yeast.

Meister P, Poidevin M, Francesconi S, Tratner I, Zarzov P, Baldacci G.

Nucleic Acids Res. 2003 Sep 1;31(17):5064-73.

9.

DNA processing is not required for ATM-mediated telomere damage response after TRF2 deletion.

Celli GB, de Lange T.

Nat Cell Biol. 2005 Jul;7(7):712-8. Epub 2005 Jun 19.

PMID:
15968270
10.

Telomere shortening alters the kinetics of the DNA damage response after ionizing radiation in human cells.

Drissi R, Wu J, Hu Y, Bockhold C, Dome JS.

Cancer Prev Res (Phila). 2011 Dec;4(12):1973-81. doi: 10.1158/1940-6207.CAPR-11-0069. Epub 2011 Sep 19.

11.

Histone H2A phosphorylation controls Crb2 recruitment at DNA breaks, maintains checkpoint arrest, and influences DNA repair in fission yeast.

Nakamura TM, Du LL, Redon C, Russell P.

Mol Cell Biol. 2004 Jul;24(14):6215-30. Erratum in: Mol Cell Biol. 2004 Sep;24(17):7820.

12.

DNA-PKcs plays a dominant role in the regulation of H2AX phosphorylation in response to DNA damage and cell cycle progression.

An J, Huang YC, Xu QZ, Zhou LJ, Shang ZF, Huang B, Wang Y, Liu XD, Wu DC, Zhou PK.

BMC Mol Biol. 2010 Mar 6;11:18. doi: 10.1186/1471-2199-11-18.

13.
14.
15.

Telomeres, histone code, and DNA damage response.

Misri S, Pandita S, Kumar R, Pandita TK.

Cytogenet Genome Res. 2008;122(3-4):297-307. doi: 10.1159/000167816. Epub 2009 Jan 30. Review.

16.

Regulation of telomere length by checkpoint genes in Schizosaccharomyces pombe.

Dahlen M, Olsson T, Kanter-Smoler G, Ramne A, Sunnerhagen P.

Mol Biol Cell. 1998 Mar;9(3):611-21.

17.
18.

Dial 9-1-1 for DNA damage: the Rad9-Hus1-Rad1 (9-1-1) clamp complex.

Parrilla-Castellar ER, Arlander SJ, Karnitz L.

DNA Repair (Amst). 2004 Aug-Sep;3(8-9):1009-14. Review.

PMID:
15279787
19.

Artemis is a phosphorylation target of ATM and ATR and is involved in the G2/M DNA damage checkpoint response.

Zhang X, Succi J, Feng Z, Prithivirajsingh S, Story MD, Legerski RJ.

Mol Cell Biol. 2004 Oct;24(20):9207-20.

20.

An imperfect G2M checkpoint contributes to chromosome instability following irradiation of S and G2 phase cells.

Krempler A, Deckbar D, Jeggo PA, Löbrich M.

Cell Cycle. 2007 Jul 15;6(14):1682-6. Epub 2007 May 21. Review.

PMID:
17637566
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