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Items: 1 to 20 of 111


The anti-invasive activity of cyclooxygenase inhibitors is regulated by the transcription factor ATF3 (activating transcription factor 3).

Bottone FG Jr, Moon Y, Kim JS, Alston-Mills B, Ishibashi M, Eling TE.

Mol Cancer Ther. 2005 May;4(5):693-703.


Transcriptional regulation of activating transcription factor 3 involves the early growth response-1 gene.

Bottone FG Jr, Moon Y, Alston-Mills B, Eling TE.

J Pharmacol Exp Ther. 2005 Nov;315(2):668-77. Epub 2005 Aug 3.


Gene modulation by the cyclooxygenase inhibitor, sulindac sulfide, in human colorectal carcinoma cells: possible link to apoptosis.

Bottone FG Jr, Martinez JM, Collins JB, Afshari CA, Eling TE.

J Biol Chem. 2003 Jul 11;278(28):25790-801. Epub 2003 May 6.


Nonsteroidal anti-inflammatory drug sulindac sulfide suppresses structural protein Nesprin-2 expression in colorectal cancer cells.

Liggett JL, Choi CK, Donnell RL, Kihm KD, Kim JS, Min KW, Noegel AA, Baek SJ.

Biochim Biophys Acta. 2014 Jan;1840(1):322-31. doi: 10.1016/j.bbagen.2013.09.032. Epub 2013 Sep 27.


The dietary compounds resveratrol and genistein induce activating transcription factor 3 while suppressing inhibitor of DNA binding/differentiation-1.

Bottone FG Jr, Alston-Mills B.

J Med Food. 2011 Jun;14(6):584-93. doi: 10.1089/jmf.2010.0110. Epub 2011 May 9.


The cyclooxygenase inhibitor sulindac sulfide inhibits EP4 expression and suppresses the growth of glioblastoma cells.

Kambe A, Yoshioka H, Kamitani H, Watanabe T, Baek SJ, Eling TE.

Cancer Prev Res (Phila). 2009 Dec;2(12):1088-99. doi: 10.1158/1940-6207.CAPR-09-0140. Epub 2009 Nov 24.


Growth inhibition and induction of apoptosis in colorectal tumor cells by cyclooxygenase inhibitors.

Richter M, Weiss M, Weinberger I, F├╝rstenberger G, Marian B.

Carcinogenesis. 2001 Jan;22(1):17-25.


PPARgamma-mediated antineoplastic effect of NSAID sulindac on human oral squamous carcinoma cells.

Nikitakis NG, Hebert C, Lopes MA, Reynolds MA, Sauk JJ.

Int J Cancer. 2002 Apr 20;98(6):817-23.


Cyclooxygenase inhibitors induce apoptosis in sinonasal cancer cells by increased expression of nonsteroidal anti-inflammatory drug-activated gene.

Kim JH, Chang JH, Rhee KH, Yoon JH, Kwon SH, Song K, Lee KW, Cho CI, Jeon JH, Kim KS.

Int J Cancer. 2008 Apr 15;122(8):1765-73.


The oncogenic effects of constitutive Stat3 signaling in salivary gland cancer cells are mediated by survivin and modulated by the NSAID sulindac.

Nikitakis NG, Scheper MA, Papanikolaou VS, Sauk JJ.

Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 2009 Jun;107(6):826-36. doi: 10.1016/j.tripleo.2008.12.054. Epub 2009 Mar 9. Retraction in: Nikitakis NG, Scheper MA, Papanikolaou VS, Sauk JJ. Oral Surg Oral Med Oral Pathol Oral Radiol. 2014 Dec;118(6):746.


The cyclooxygenase inhibitor indomethacin modulates gene expression and represses the extracellular matrix protein laminin gamma1 in human glioblastoma cells.

Ishibashi M, Bottone FG Jr, Taniura S, Kamitani H, Watanabe T, Eling TE.

Exp Cell Res. 2005 Jan 15;302(2):244-52.


Gene modulation by Cox-1 and Cox-2 specific inhibitors in human colorectal carcinoma cancer cells.

Bottone FG Jr, Martinez JM, Alston-Mills B, Eling TE.

Carcinogenesis. 2004 Mar;25(3):349-57. Epub 2003 Nov 21.


Colon tumor cell growth-inhibitory activity of sulindac sulfide and other nonsteroidal anti-inflammatory drugs is associated with phosphodiesterase 5 inhibition.

Tinsley HN, Gary BD, Thaiparambil J, Li N, Lu W, Li Y, Maxuitenko YY, Keeton AB, Piazza GA.

Cancer Prev Res (Phila). 2010 Oct;3(10):1303-13. doi: 10.1158/1940-6207.CAPR-10-0030. Epub 2010 Sep 28.


GATA-6 transcriptional regulation of 15-lipoxygenase-1 during NSAID-induced apoptosis in colorectal cancer cells.

Shureiqi I, Jiang W, Fischer SM, Xu X, Chen D, Lee JJ, Lotan R, Lippman SM.

Cancer Res. 2002 Feb 15;62(4):1178-83.

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