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Items: 1 to 20 of 108

1.

The role of the Shigella flexneri yihE gene in LPS synthesis and virulence.

Edwards-Jones B, Langford PR, Kroll JS, Yu J.

Microbiology. 2004 Apr;150(Pt 4):1079-84.

PMID:
15073317
2.
3.

Influence of the yihE gene of Shigella flexneri on global gene expression: on analysis using DNA arrays.

Li MS, Kroll JS, Yu J.

Biochem Biophys Res Commun. 2001 Oct 19;288(1):91-100.

PMID:
11594757
4.

Outer membrane protein A (OmpA): a new player in shigella flexneri protrusion formation and inter-cellular spreading.

Ambrosi C, Pompili M, Scribano D, Zagaglia C, Ripa S, Nicoletti M.

PLoS One. 2012;7(11):e49625. doi: 10.1371/journal.pone.0049625. Epub 2012 Nov 14.

5.

Construction, detection and microarray analysis on the Shigella flexneri 2a sitC mutant.

Liu M, Liu H, Sun L, Dong J, Xue Y, Chen S, Jin Q.

Sci China C Life Sci. 2005 Jun;48(3):228-40.

PMID:
16092755
6.

Avirulence of rough mutants of Shigella flexneri: requirement of O antigen for correct unipolar localization of IcsA in the bacterial outer membrane.

Sandlin RC, Lampel KA, Keasler SP, Goldberg MB, Stolzer AL, Maurelli AT.

Infect Immun. 1995 Jan;63(1):229-37.

7.

The regulatory protein PhoP controls susceptibility to the host inflammatory response in Shigella flexneri.

Moss JE, Fisher PE, Vick B, Groisman EA, Zychlinsky A.

Cell Microbiol. 2000 Dec;2(6):443-52.

PMID:
11207599
8.

A DNA adenine methylase mutant of Shigella flexneri shows no significant attenuation of virulence.

Honma Y, Fernández RE, Maurelli AT.

Microbiology. 2004 Apr;150(Pt 4):1073-8.

PMID:
15073316
9.
10.

Two msbB genes encoding maximal acylation of lipid A are required for invasive Shigella flexneri to mediate inflammatory rupture and destruction of the intestinal epithelium.

D'Hauteville H, Khan S, Maskell DJ, Kussak A, Weintraub A, Mathison J, Ulevitch RJ, Wuscher N, Parsot C, Sansonetti PJ.

J Immunol. 2002 May 15;168(10):5240-51.

11.

Deletion of pic results in decreased virulence for a clinical isolate of Shigella flexneri 2a from China.

Zhang J, Qian L, Wu Y, Cai X, Li X, Cheng X, Qu D.

BMC Microbiol. 2013 Feb 8;13:31. doi: 10.1186/1471-2180-13-31.

12.

Absence of O antigen suppresses Shigella flexneri IcsA autochaperone region mutations.

Teh MY, Tran EN, Morona R.

Microbiology. 2012 Nov;158(Pt 11):2835-50. doi: 10.1099/mic.0.062471-0. Epub 2012 Aug 30.

PMID:
22936034
13.
14.

Optimization of virulence functions through glucosylation of Shigella LPS.

West NP, Sansonetti P, Mounier J, Exley RM, Parsot C, Guadagnini S, Prévost MC, Prochnicka-Chalufour A, Delepierre M, Tanguy M, Tang CM.

Science. 2005 Feb 25;307(5713):1313-7.

15.

Shigella flexneri cell-to-cell spread, and growth and inflammation in mice, is limited by the outer membrane protease IcsP.

Tran EN, Attridge SR, Teh MY, Morona R.

FEMS Microbiol Lett. 2015 Jun;362(12):fnv088. doi: 10.1093/femsle/fnv088. Epub 2015 May 29.

16.

Enteroinvasive Escherichia coli vs. Shigella flexneri: how different patterns of gene expression affect virulence.

Moreno AC, Ferreira LG, Martinez MB.

FEMS Microbiol Lett. 2009 Dec;301(2):156-63. doi: 10.1111/j.1574-6968.2009.01815.x. Epub 2009 Oct 7.

17.

Regulation of O-antigen chain length is required for Shigella flexneri virulence.

Van den Bosch L, Manning PA, Morona R.

Mol Microbiol. 1997 Feb;23(4):765-75.

18.

IcsA is a Shigella flexneri adhesin regulated by the type III secretion system and required for pathogenesis.

Brotcke Zumsteg A, Goosmann C, Brinkmann V, Morona R, Zychlinsky A.

Cell Host Microbe. 2014 Apr 9;15(4):435-45. doi: 10.1016/j.chom.2014.03.001.

19.

dksA is required for intercellular spread of Shigella flexneri via an RpoS-independent mechanism.

Mogull SA, Runyen-Janecky LJ, Hong M, Payne SM.

Infect Immun. 2001 Sep;69(9):5742-51.

20.

IcsA surface presentation in Shigella flexneri requires the periplasmic chaperones DegP, Skp, and SurA.

Purdy GE, Fisher CR, Payne SM.

J Bacteriol. 2007 Aug;189(15):5566-73. Epub 2007 May 25.

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