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Results: 1 to 20 of 120

Similar articles for PubMed (Select 11160159)

1.

Inhibition of STAT3 signaling leads to apoptosis of leukemic large granular lymphocytes and decreased Mcl-1 expression.

Epling-Burnette PK, Liu JH, Catlett-Falcone R, Turkson J, Oshiro M, Kothapalli R, Li Y, Wang JM, Yang-Yen HF, Karras J, Jove R, Loughran TP Jr.

J Clin Invest. 2001 Feb;107(3):351-62.

2.

Inhibition of STAT3 signaling induces apoptosis and decreases survivin expression in primary effusion lymphoma.

Aoki Y, Feldman GM, Tosato G.

Blood. 2003 Feb 15;101(4):1535-42. Epub 2002 Oct 3.

3.

Cooperative regulation of Mcl-1 by Janus kinase/stat and phosphatidylinositol 3-kinase contribute to granulocyte-macrophage colony-stimulating factor-delayed apoptosis in human neutrophils.

Epling-Burnette PK, Zhong B, Bai F, Jiang K, Bailey RD, Garcia R, Jove R, Djeu JY, Loughran TP Jr, Wei S.

J Immunol. 2001 Jun 15;166(12):7486-95.

4.

Serine phosphorylation of STAT3 is essential for Mcl-1 expression and macrophage survival.

Liu H, Ma Y, Cole SM, Zander C, Chen KH, Karras J, Pope RM.

Blood. 2003 Jul 1;102(1):344-52. Epub 2003 Mar 13.

6.

Inhibition of constitutively active Stat3 suppresses growth of human ovarian and breast cancer cells.

Burke WM, Jin X, Lin HJ, Huang M, Liu R, Reynolds RK, Lin J.

Oncogene. 2001 Nov 29;20(55):7925-34.

8.

Inhibition of constitutively activated Stat3 correlates with altered Bcl-2/Bax expression and induction of apoptosis in mycosis fungoides tumor cells.

Nielsen M, Kaestel CG, Eriksen KW, Woetmann A, Stokkedal T, Kaltoft K, Geisler C, Röpke C, Odum N.

Leukemia. 1999 May;13(5):735-8.

PMID:
10374878
9.
11.

Constitutive activation of Stat3 by the Src and JAK tyrosine kinases participates in growth regulation of human breast carcinoma cells.

Garcia R, Bowman TL, Niu G, Yu H, Minton S, Muro-Cacho CA, Cox CE, Falcone R, Fairclough R, Parsons S, Laudano A, Gazit A, Levitzki A, Kraker A, Jove R.

Oncogene. 2001 May 3;20(20):2499-513.

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14.

Stat3 activation by Src induces specific gene regulation and is required for cell transformation.

Turkson J, Bowman T, Garcia R, Caldenhoven E, De Groot RP, Jove R.

Mol Cell Biol. 1998 May;18(5):2545-52.

15.

The JAK-inhibitor, JAB/SOCS-1 selectively inhibits cytokine-induced, but not v-Src induced JAK-STAT activation.

Iwamoto T, Senga T, Naito Y, Matsuda S, Miyake Y, Yoshimura A, Hamaguchi M.

Oncogene. 2000 Sep 28;19(41):4795-801.

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17.

Analysis of signal transducer and activator of transcription 3 (STAT3) in gastrointestinal stromal tumors.

Paner GP, Silberman S, Hartman G, Micetich KC, Aranha GV, Alkan S.

Anticancer Res. 2003 May-Jun;23(3B):2253-60.

PMID:
12894500
18.

Constitutive activation of Stat3 signaling confers resistance to apoptosis in human U266 myeloma cells.

Catlett-Falcone R, Landowski TH, Oshiro MM, Turkson J, Levitzki A, Savino R, Ciliberto G, Moscinski L, Fernández-Luna JL, Nuñez G, Dalton WS, Jove R.

Immunity. 1999 Jan;10(1):105-15.

19.

Phosphatidylinositol-3-phosphate kinase pathway activation protects leukemic large granular lymphocytes from undergoing homeostatic apoptosis.

Schade AE, Powers JJ, Wlodarski MW, Maciejewski JP.

Blood. 2006 Jun 15;107(12):4834-40. Epub 2006 Feb 16.

20.

Granulocyte-macrophage colony-stimulating factor (GM-CSF) induces antiapoptotic and proapoptotic signals in acute myeloid leukemia.

Faderl S, Harris D, Van Q, Kantarjian HM, Talpaz M, Estrov Z.

Blood. 2003 Jul 15;102(2):630-7. Epub 2003 Mar 27.

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