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Similar articles for PubMed (Select 10899918)

1.

N-Acetylaspartylglutamate: the most abundant peptide neurotransmitter in the mammalian central nervous system.

Neale JH, Bzdega T, Wroblewska B.

J Neurochem. 2000 Aug;75(2):443-52. Review.

PMID:
10899918
2.
3.

NAAG inhibits KCl-induced [(3)H]-GABA release via mGluR3, cAMP, PKA and L-type calcium conductance.

Zhao J, Ramadan E, Cappiello M, Wroblewska B, Bzdega T, Neale JH.

Eur J Neurosci. 2001 Jan;13(2):340-6.

PMID:
11168538
4.

beta-NAAG rescues LTP from blockade by NAAG in rat dentate gyrus via the type 3 metabotropic glutamate receptor.

Lea PM 4th, Wroblewska B, Sarvey JM, Neale JH.

J Neurophysiol. 2001 Mar;85(3):1097-106.

5.

Effects of N-acetylaspartylglutamate (NAAG) at group II mGluRs and NMDAR.

Fricker AC, Mok MH, de la Flor R, Shah AJ, Woolley M, Dawson LA, Kew JN.

Neuropharmacology. 2009 May-Jun;56(6-7):1060-7. doi: 10.1016/j.neuropharm.2009.03.002. Epub 2009 Mar 12.

PMID:
19285517
6.

The neuroactive peptide N-acetylaspartylglutamate is not an agonist at the metabotropic glutamate receptor subtype 3 of metabotropic glutamate receptor.

Chopra M, Yao Y, Blake TJ, Hampson DR, Johnson EC.

J Pharmacol Exp Ther. 2009 Jul;330(1):212-9. doi: 10.1124/jpet.109.152553. Epub 2009 Apr 23.

7.

Immunohistological and electrophysiological evidence that N-acetylaspartylglutamate is a co-transmitter at the vertebrate neuromuscular junction.

Walder KK, Ryan SB, Bzdega T, Olszewski RT, Neale JH, Lindgren CA.

Eur J Neurosci. 2013 Jan;37(1):118-29. doi: 10.1111/ejn.12027. Epub 2012 Nov 8.

8.

N-acetylaspartylglutamate stimulates metabotropic glutamate receptor 3 to regulate expression of the GABA(A) alpha6 subunit in cerebellar granule cells.

Ghose S, Wroblewska B, Corsi L, Grayson DR, De Blas AL, Vicini S, Neale JH.

J Neurochem. 1997 Dec;69(6):2326-35.

PMID:
9375663
9.
10.

Advances in understanding the peptide neurotransmitter NAAG and appearance of a new member of the NAAG neuropeptide family.

Neale JH, Olszewski RT, Zuo D, Janczura KJ, Profaci CP, Lavin KM, Madore JC, Bzdega T.

J Neurochem. 2011 Aug;118(4):490-8. doi: 10.1111/j.1471-4159.2011.07338.x. Epub 2011 Jul 1. Review.

11.

N-acetylaspartylglutamate and beta-NAAG protect against injury induced by NMDA and hypoxia in primary spinal cord cultures.

Yourick DL, Koenig ML, Durden AV, Long JB.

Brain Res. 2003 Nov 21;991(1-2):56-64.

PMID:
14575876
12.
13.

N-acetylaspartylglutamate selectively activates mGluR3 receptors in transfected cells.

Wroblewska B, Wroblewski JT, Pshenichkin S, Surin A, Sullivan SE, Neale JH.

J Neurochem. 1997 Jul;69(1):174-81.

PMID:
9202308
14.

NAAG fails to antagonize synaptic and extrasynaptic NMDA receptors in cerebellar granule neurons.

Losi G, Vicini S, Neale J.

Neuropharmacology. 2004 Mar;46(4):490-6.

PMID:
14975672
15.

Central N-acetyl aspartylglutamate deficit: a possible pathogenesis of schizophrenia.

Tsai SJ.

Med Sci Monit. 2005 Sep;11(9):HY39-45. Epub 2005 Aug 26. Review.

PMID:
16127367
16.

The regional distribution of N-acetylaspartylglutamate (NAAG) and peptidase activity against NAAG in the rat nervous system.

Fuhrman S, Palkovits M, Cassidy M, Neale JH.

J Neurochem. 1994 Jan;62(1):275-81.

PMID:
8263527
17.

The neurotransmitter N-acetylaspartylglutamate in models of pain, ALS, diabetic neuropathy, CNS injury and schizophrenia.

Neale JH, Olszewski RT, Gehl LM, Wroblewska B, Bzdega T.

Trends Pharmacol Sci. 2005 Sep;26(9):477-84. Review.

PMID:
16055199
18.

N-Acetylated alpha-linked acidic dipeptidase converts N-acetylaspartylglutamate from a neuroprotectant to a neurotoxin.

Thomas AG, Vornov JJ, Olkowski JL, Merion AT, Slusher BS.

J Pharmacol Exp Ther. 2000 Oct;295(1):16-22.

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20.

Intracerebroventricular administration of N-acetylaspartylglutamate (NAAG) peptidase inhibitors is analgesic in inflammatory pain.

Yamamoto T, Kozikowski A, Zhou J, Neale JH.

Mol Pain. 2008 Aug 1;4:31. doi: 10.1186/1744-8069-4-31.

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