Inhibition of brain protein kinase C subtypes by lead

J Pharmacol Exp Ther. 1993 Feb;264(2):757-61.

Abstract

Protein kinase C (PKC) is an important enzyme in mediating cellular signal transduction and neuronal plasticity. Extremely low concentrations (picomolar range) of Pb++ have been reported to activate partially purified PKC from rat brain (Markovac and Goldstein, 1988). However, the lead activation of PKC at such low concentrations is still a matter of discussion (Simons, 1989). To clarify this point, we have examined the lead effect on highly purified PKC subtypes. Pb++ was found to be a potent inhibitor for all three PKC subtypes (types I, II and III) with IC50 of 2 to 10 microM. Characterization of this lead inhibition of PKC suggests that 1) the inhibition is not due to the competition with Ca++, 2) the site of action of lead is on the catalytic domain of PKC, 3) the inhibition is not dependent on the mode of activation (phosphatidylserine/diacylglycerol vs. cis-unsaturated fatty acid) and 4) the inhibition is totally reversible.

MeSH terms

  • Animals
  • Brain / drug effects
  • Brain / enzymology*
  • Calcium / pharmacology
  • In Vitro Techniques
  • Lead / toxicity*
  • Male
  • Protein Kinase C / antagonists & inhibitors*
  • Rats
  • Rats, Sprague-Dawley

Substances

  • Lead
  • Protein Kinase C
  • Calcium