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Arch Ophthalmol. 1993 Sep;111(9):1229-33.

Central serous chorioretinopathy in endogenous hypercortisolism.

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  • 1Ophthalmic Genetics and Clinical Services Branch, National Eye Institute, National Institutes of Health, Bethesda, Md.

Abstract

OBJECTIVE:

To examine the potential association of central serous chorioretinopathy with endogenous hypercortisolism (Cushing's syndrome).

DESIGN:

Ophthalmologic survey of consecutively admitted patients with endogenous Cushing's syndrome.

SETTING:

An eye clinic of a research center (National Eye Institute, Bethesda, Md).

PATIENTS:

Sixty consecutive patients with confirmed endogenous Cushing's syndrome.

MAIN OUTCOME MEASURES:

Findings from complete ophthalmologic evaluation.

RESULTS:

Three (5%) of 60 patients had one or more episodes of appropriately documented central serous chorioretinopathy. In all cases, the episodes occurred during the period of hypercortisolism.

CONCLUSIONS:

Central serous chorioretinopathy is an uncommon manifestation of endogenous Cushing's syndrome. Since central serous chorioretinopathy has been associated with other hypercortisolemic states, we suggest that glucocorticoids may play a role in the development of this disease.

PMID:
8363466
[PubMed - indexed for MEDLINE]
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