Putative functional SNPs in SLC22A3 and H3F3B might influence the development of CAD by regulating the lipid levels

Thromb Res. 2018 Aug:168:37-39. doi: 10.1016/j.thromres.2018.06.002. Epub 2018 Jun 2.
No abstract available

Keywords: Coronary artery disease; Functional variants; Gene expression; Genome-wide association study; Low-density lipoprotein cholesterol; Total cholesterol; m(6)A.

Publication types

  • Letter

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Cholesterol, LDL / blood
  • Cholesterol, LDL / genetics
  • Coronary Artery Disease / blood*
  • Coronary Artery Disease / etiology
  • Coronary Artery Disease / genetics*
  • Female
  • Genetic Predisposition to Disease
  • Histones / blood
  • Histones / genetics*
  • Humans
  • Lipids / blood*
  • Male
  • Membrane Proteins / biosynthesis
  • Membrane Proteins / genetics
  • Middle Aged
  • Organic Cation Transport Proteins / blood
  • Organic Cation Transport Proteins / genetics*
  • Polymorphism, Single Nucleotide
  • Risk Factors
  • Young Adult

Substances

  • Cholesterol, LDL
  • H3-3A protein, human
  • Histones
  • Lipids
  • Membrane Proteins
  • Organic Cation Transport Proteins
  • UBXN4 protein, human
  • solute carrier family 22 (organic cation transporter), member 3