Ann Neurol. 1989 Sep;26(3):392-5.

Hyperkalemic periodic paralysis in Gordon's syndrome: a possible defect in atrial natriuretic peptide function.

Pasman JW, Gabreëls FJ, Semmekrot B, Renier WO, Monnens LA.

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Institute of Neurology, University Hospital Nijmegen, The Netherlands.

Abstract

We present the case of a 14-year-old boy who had secondary hyperkalemic periodic paralysis caused by Gordon's syndrome. This syndrome consists of hypertension, tubular acidosis, and hyperkalemia with normal glomerular filtration rate. The pathophysiological mechanism is still unknown. Pathophysiological studies suggest that in this disorder the kidney lacks sensitivity to atrial natriuretic peptide. After treatment with hydrochlorothiazide, serum potassium and plasma aldosterone values, plasma renin activity, and blood pressure became normal and the attacks of periodic paralysis disappeared.

PMID:: 2529811; [PubMed - indexed for MEDLINE]

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