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Oncoimmunology. 2012 Oct 1;1(7):1200-1201.

Opposing consequences of signaling through EGF family members: Escape from CTLs could be a bait for NK cells.

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  • 1Department of Oncology and Pathology; Immune and Gene Therapy Laboratory; Cancer Center Karolinska; Karolinska Institutet; Stockholm, Sweden.

Abstract

Oncogenes have been traditionally viewed as molecular drivers for tumor growth and survival. Recent evidence indicates that oncogenes may facilitate the escape of malignant cells from immune recognition and elimination. In this article, we discuss the implications of the overexpression of epidermal growth factor receptor (EGFR) family members on immune escape of tumors and immunotherapy.

PMID:
23170279
[PubMed]
PMCID:
PMC3494645
Free PMC Article
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