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    J Virol. 2012 Apr;86(7):4029-33. Epub 2012 Jan 25.

    Depletion of intracellular zinc inhibits the ubiquitin ligase activity of viral regulatory protein ICP0 and restricts herpes simplex virus 1 replication in cell culture.

    Source

    MRC—University of Glasgow Centre for Virus Research, Glasgow, Scotland, United Kingdom.

    Abstract

    The viral ubiquitin ligase ICP0 stimulates the onset of HSV-1 lytic infection and productive reactivation of viral genomes from latency. In order to mediate these processes, it requires its C3HC4 RING finger domain, a tertiary structural fold that is coordinated by the binding of two zinc (Zn(2+)) atoms. Here we formally demonstrate that Zn(2+) binding and intracellular Zn(2+) levels are critical for ICP0's biochemical activity and that depletion of intracellular Zn(2+) severely attenuates HSV-1 replication.

    PMID:
    22278229
    [PubMed - in process]
    PMCID:
    PMC3302540
    [Available on 2012/10/1]

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