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Congenit Heart Dis. 2012 May-Jun;7(3):E10-3. doi: 10.1111/j.1747-0803.2011.00557.x. Epub 2011 Jul 31.

Recurrent protein-losing enteropathy and tricuspid valve insufficiency in a transplanted heart: a causal relationship?

Author information

  • 1Division of Pediatric Cardiology Department of Cardiovascular Surgery, Children's Hospital of Michigan, 3901 Beaubien Blvd., Detroit, MI 48201, USA. ssanjeev@dmc.org

Abstract

This case report describes a toddler who developed a protein-losing enteropathy (PLE) 4 years after orthotopic heart transplantation (OHT). He was born with a hypoplastic left heart syndrome for which he underwent a successful Norwood procedure, a Hemi-Fontan palliation, and a Fontan palliation at 18 months of age. Fifteen months following the Fontan operation, he developed a PLE and Fontan failure requiring OHT. Four years after OHT, he developed a severe tricuspid regurgitation and a PLE. His PLE improved after tricuspid valve replacement. It is now 2 years since his tricuspid valve replacement and he remains clinically free of ascites and peripheral edema with a normal serum albumin level. His prosthetic tricuspid valve is functioning normally.

© 2011 Wiley Periodicals, Inc.

PMID:
21801319
[PubMed - indexed for MEDLINE]
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