PLoS Biol. 2011 Jan 18;9(1):e1001008.

The importance of synthetic associations will only be resolved empirically.

Center for Human Genome Variation, Duke University School of Medicine, Durham, North Carolina, United States of America. d.goldstein@duke.edu

Supplemental Content

Synthetic associations are unlikely to account for many common disease genome-wide association signals. [PLoS Biol. 2011]
Synthetic associations are unlikely to account for many common disease genome-wide association signals.
Anderson CA, Soranzo N, Zeggini E, Barrett JC. PLoS Biol. 2011 Jan 18; 9(1):e1000580. Epub 2011 Jan 18.
Review Synthetic associations in the context of genome-wide association scan signals. [Hum Mol Genet. 2010]
Review Synthetic associations in the context of genome-wide association scan signals.
Orozco G, Barrett JC, Zeggini E. Hum Mol Genet. 2010 Oct 15; 19(R2):R137-44. Epub 2010 Aug 30.
Common disease: are causative alleles common or rare? [PLoS Biol. 2011]
Common disease: are causative alleles common or rare?
Shields R. PLoS Biol. 2011 Jan 18; 9(1):e1001009. Epub 2011 Jan 18.
Synthetic associations created by rare variants do not explain most GWAS results. [PLoS Biol. 2011]
Synthetic associations created by rare variants do not explain most GWAS results.
Wray NR, Purcell SM, Visscher PM. PLoS Biol. 2011 Jan 18; 9(1):e1000579. Epub 2011 Jan 18.
Review Human genetic variation and its contribution to complex traits. [Nat Rev Genet. 2009]
Review Human genetic variation and its contribution to complex traits.
Frazer KA, Murray SS, Schork NJ, Topol EJ. Nat Rev Genet. 2009 Apr; 10(4):241-51.

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