Neuropsychopharmacology. 2010 Mar;35(4):853-4. doi: 10.1038/npp.2009.205.

An animal model for the treatment of Alzheimer's disease: potential for a new direction in therapies that enhance the epigenic mechanisms of memory formation.

Eichenbaum HB.

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Center for Memory and Brain, Boston University, Boston, MA 02115, USA. hbe@bu.edu

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Inhibitors of class 1 histone deacetylases reverse contextual memory deficits in a mouse model of Alzheimer's disease. [Neuropsychopharmacology. 2010]
Inhibitors of class 1 histone deacetylases reverse contextual memory deficits in a mouse model of Alzheimer's disease.Kilgore M, Miller CA, Fass DM, Hennig KM, Haggarty SJ, Sweatt JD, Rumbaugh G. Neuropsychopharmacology. 2010 Mar; 35(4):870-80. Epub 2009 Dec 9.

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PMCID:: PMC3055360

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An animal model for the treatment of Alzheimer's disease: potential for a new di...
An animal model for the treatment of Alzheimer's disease: potential for a new direction in therapies that enhance the epigenic mechanisms of memory formation.
Neuropsychopharmacology. 2010 Mar ;35(4):853-4. doi: 10.1038/npp.2009.205.

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