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    Results: 4

    1.

    Investigations on the role of base excision repair and non-homologous end-joining pathways in sodium selenite-induced toxicity and mutagenicity in Saccharomyces cerevisiae.

    Mániková D, Vlasáková D, Loduhová J, Letavayová L, Vigasová D, Krascsenitsová E, Vlcková V, Brozmanová J, Chovanec M.

    Mutagenesis. 2010 Mar;25(2):155-62. Epub 2009 Dec 2.

    PMID:
    19955329
    [PubMed - indexed for MEDLINE]
    Free Article
    2.

    Abrogation of the Chk1-Pds1 checkpoint leads to tolerance of persistent single-strand breaks in Saccharomyces cerevisiae.

    Karumbati AS, Wilson TE.

    Genetics. 2005 Apr;169(4):1833-44. Epub 2005 Jan 31.

    PMID:
    15687272
    [PubMed - indexed for MEDLINE]
    Free PMC Article
    3.

    The role of yeast DNA 3'-phosphatase Tpp1 and rad1/Rad10 endonuclease in processing spontaneous and induced base lesions.

    Karumbati AS, Deshpande RA, Jilani A, Vance JR, Ramotar D, Wilson TE.

    J Biol Chem. 2003 Aug 15;278(33):31434-43. Epub 2003 Jun 3.

    PMID:
    12783866
    [PubMed - indexed for MEDLINE]
    Free Article
    4.

    Repair of DNA strand breaks by the overlapping functions of lesion-specific and non-lesion-specific DNA 3' phosphatases.

    Vance JR, Wilson TE.

    Mol Cell Biol. 2001 Nov;21(21):7191-8.

    PMID:
    11585902
    [PubMed - indexed for MEDLINE]
    Free PMC Article

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