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Raf-1: a kinase currently without a cause but not lacking in effects.
Division of Cellular and Molecular Biology, Dana-Farber Cancer Institute, Boston, Massachusetts.
PMID: 1846778 [PubMed - indexed for MEDLINE]
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Cited by 31 PubMed Central articles
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Raf-induced proliferation or cell cycle arrest is determined by the level of Raf activity with arrest mediated by p21Cip1.
Woods D, Parry D, Cherwinski H, Bosch E, Lees E, McMahon M.
Mol Cell Biol. 1997 Sep; 17(9):5598-611.
[Mol Cell Biol. 1997]
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The Ras-Raf pathway is activated in human immunodeficiency virus-infected monocytes and particpates in the activation of NF-kappa B.
Folgueira L, Algeciras A, MacMorran WS, Bren GD, Paya CV.
J Virol. 1996 Apr; 70(4):2332-8.
[J Virol. 1996]
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Thrombin receptor activation elicits rapid protein tyrosine phosphorylation and stimulation of the raf-1/MAP kinase pathway preceding delayed mitogenesis in cultured rat aortic smooth muscle cells: evidence for an obligate autocrine mechanism promoting cell proliferation induced by G-protein-coupled receptor agonist.
Molloy CJ, Pawlowski JE, Taylor DS, Turner CE, Weber H, Peluso M.
J Clin Invest. 1996 Mar 1; 97(5):1173-83.
[J Clin Invest. 1996]
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