Display Settings:

Format

Send to:

Choose Destination
    J Acquir Immune Defic Syndr. 2008 Jun 1;48(2):149-55.

    Early changes in T-cell activation predict antiretroviral success in salvage therapy of HIV infection.

    Source

    Mayo Clinic, Rochester, MN 55905, USA.

    Abstract

    OBJECTIVE:

    Because effective antiretroviral therapy (ART) reduces immune activation, we hypothesize that early changes in immune activation are associated with subsequent virologic response to therapy.

    DESIGN:

    Observational cohort study.

    SETTING:

    Institutional HIV clinic.

    SUBJECTS:

    Thirty-four adult HIV patients with virologic failure on their current antiretroviral regimen.

    INTERVENTION:

    Change to salvage regimen selected by patient's physician.

    MAIN OUTCOME MEASURES:

    Measures of immune activation at baseline and at 2, 4, 8, and 24 weeks after enrollment. Data were analyzed by proportional hazards (PH) models.

    RESULTS:

    PH models showed that reductions between baseline and week 2 in expression of CD38 (P = 0.02) or CD95 (P = 0.02) on CD4 T cells were associated with increased likelihood of achieving virologic suppression. Kaplan-Meier analysis demonstrated that patients who had reductions within the first 2 weeks of therapy in CD4 T-cell expression of CD38 (P = 0.003) or CD95 (P = 0.08) were more likely to achieve viral suppression than those who did not.

    CONCLUSIONS:

    Reduced CD4 T-cell expression of CD38 and CD95 occurring within 2 weeks of salvage therapy is associated with subsequent viral suppression. Monitoring CD38 and CD95 may allow earlier assessment of the response to ART.

    PMID:
    18360289
    [PubMed - indexed for MEDLINE]
    PMCID: PMC3149796
    Free PMC Article

    Images from this publication.See all images (1) Free text

    FIGURE 1

      Supplemental Content

      Click here to read

      Recent activity

      Your browsing activity is empty.

      Activity recording is turned off.

      Turn recording back on

      See more...
      Write to the Help Desk