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    Br J Pharmacol. 2007 Mar;150(5):538-40. Epub 2007 Jan 22.

    Cardiovascular protection with sildenafil following chronic inhibition of nitric oxide synthase.

    Source

    Department of Internal Medicine, Division of Cardiology, Virginia Commonwealth University, Richmond, VA 23298, USA. rakesh@hsc.vcu.edu

    Abstract

    During the past 18 years, sildenafil has evolved from a potential anti-angina drug to an on-demand treatment for erectile dysfunction and more recently to a new orally active treatment for pulmonary hypertension. Recent studies suggest that the drug has powerful cardioprotective effect against ischemia/reperfusion injury, doxorubicin-induced cardiomyopathy and anti-hypertensive effect induced by chronic inhibition of nitric oxide synthase in animals. Based on several recent basic and clinical studies, it is clear that sildenafil and other clinically approved type-5 phosphodiesterase-5 inhibitors including vardenafil and tadalafil will eventually be developed for several cardiovascular indications including essential hypertension, endothelial dysfunction, ischemia/reperfusion injury, myocardial infarction, ventricular remodeling and heart failure.

    PMID:
    17245364
    [PubMed - indexed for MEDLINE]
    PMCID:
    PMC2189762
    Free PMC Article

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