Ann Allergy. 1991 Apr;66(4):335-8.

Proteins of the complement system and acute phase reactants in sera of patients with spinal cord injury.

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Casa Colina Hospital for Rehabilitative Medicine, Pomona, California.

Abstract

Complement activity was studied in patients with spinal cord transection. In some sera acute phase reactants: haptoglobin, C-reactive proteins, ceruloplasmin, as well as fibrinogen and fibrin degradation products, and immune complexes were monitored. Complement and acute phase reactants are increased in a majority of patients. Continuing inflammation and release of inflammatory mediators could be responsible for poor healing that commonly occurs in spinal cord injury. Urinary tract and other infections are associated with some but not all of the protein abnormalities. These proinflammatory proteins may contribute to the lack of healing of spinal transection.

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[Acute phase reaction after inflammatory injury. Experimental study]. [Rev Med Univ Navarra. 1988]
[Acute phase reaction after inflammatory injury. Experimental study].
Sánchez L, Solares MC, Zozaya JM, Muñoz M, Liso P. Rev Med Univ Navarra. 1988 Jan-Mar; 32(1):47-50.
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Cited by 2 PubMed Central articles

The alternative and terminal pathways of complement mediate post-traumatic spinal cord inflammation and injury. [Am J Pathol. 2010]
The alternative and terminal pathways of complement mediate post-traumatic spinal cord inflammation and injury.
Qiao F, Atkinson C, Kindy MS, Shunmugavel A, Morgan BP, Song H, Tomlinson S. Am J Pathol. 2010 Dec; 177(6):3061-70. Epub 2010 Oct 15.
Deficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injury. [J Neurosci. 2008]
Deficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injury.
Galvan MD, Luchetti S, Burgos AM, Nguyen HX, Hooshmand MJ, Hamers FP, Anderson AJ. J Neurosci. 2008 Dec 17; 28(51):13876-88.

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Proteins of the complement system and acute phase reactants in sera of patients with spinal cord injury.

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