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    Mol Cell. 2005 Mar 18;17(6):751-2.

    Filling the gaps in replication restart pathways.

    Lovett ST.

    Rosenstiel Basic Medical Sciences Research Center, Department of Biology, Brandeis University, Waltham, MA 02454, USA. lovett@brandeis.edu

    Comment on:

    Recent work by clarifies two mechanisms of replication fork assembly in E. coli: in vitro, the presence of a leading nascent strand dictates a PriA/B DnaT-dependent mechanism, whereas a gapped fork specifies PriC-mediated restart.

    PMID: 15780929 [PubMed - indexed for MEDLINE]

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