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    Crit Care. 2005 Apr;9(2):134-5. Epub 2004 Nov 15.

    Dear vasopressin, where is your place in septic shock?

    Duenser MW, Hasibeder WR.

    Division of General and Surgical Intensive Care Medicine, Department of Anesthesiology and Critical Care Medicine, The Medical University of Innsbruck, Austria. csab3987@uibk.ac.at

    Cardiovascular failure is one of the central therapeutic problems in patients with severe infection. Although norepinephrine is a potent and, in most cases, highly effective vasopressor agent, very high dosages leading to significant side effects can be necessary to stabilize advanced shock. As a supplementary vasopressor, arginine vasopressin can reverse hemodynamic failure and significantly decrease norepinephrine dosages. Whether the promising possibility of 'bridging' advanced septic shock when the benefit/risk ratio of catecholamine therapy leaves a clinically tolerable range may improve quantitative and qualitative patient outcome can only be determined by a large, prospective, randomized study.

    PMID: 15774061 [PubMed - indexed for MEDLINE]

    PMCID: 1175913

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