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    J Neuroimmunol. 2005 Apr;161(1-2):101-12.

    A paradoxical role of APCs in the induction of intravenous tolerance in experimental autoimmune encephalomyelitis.

    Source

    Department of Neurology, Thomas Jefferson University, 300 JHN Building, 900 Walnut Street, Philadelphia, PA 19107, USA. guang-xian.zhang@jefferson.edu

    Abstract

    The central role of T cells in the induction of tolerance to autoantigens has been well documented. However, the role of antigen-presenting cells (APCs) in this process is not yet fully understood. To better understand the contribution of APCs in tolerance, we studied the interaction of purified APCs and CD4(+) T cells in a model of intravenous (i.v.) tolerance to experimental autoimmune encephalomyelitis (EAE). As expected, we found that T cells were tolerized to the autoantigen after i.v. injection. However, purified APCs obtained from MOG-i.v.-treated mice were paradoxically immuno-stimulatory, as they induced a non-specific Th1-type response both in vitro and in vivo. We conclude that blocking such APC activation would enhance the effectiveness of tolerance induction.

    PMID:
    15748949
    [PubMed - indexed for MEDLINE]

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