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Figure 1. Flow chart for the identification of SNVs in the mouse 9500 APL genome.. From: Sequencing a mouse acute promyelocytic leukemia genome reveals genetic events relevant for disease progression.
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Figure 6. Schematic representation of the 4 Kdm6a deletions found in mouse APL tumors (tumors 9500, 9520, 26, and 9495) and a KDM6A deletion found in a human AML patient (sample 750152). . From: Sequencing a mouse acute promyelocytic leukemia genome reveals genetic events relevant for disease progression.
Figure 5. Heat map of Kdm6a deletions in 4 mouse APL tumors based on targeted NimbleGen custom 12 × 135 K CGH array data. . From: Sequencing a mouse acute promyelocytic leukemia genome reveals genetic events relevant for disease progression.
Figure 2. Heat map of residual 129/SvJ SNPs by chromosomal location.. From: Sequencing a mouse acute promyelocytic leukemia genome reveals genetic events relevant for disease progression.
Figure 4. Structural variant analysis of the sequenced mouse APL genome, with discovery and validation of a somatic 150-kb deletion on chromosome X that included nearly all of the Kdm6a gene also somatically deleted in 1 human AML sample. . From: Sequencing a mouse acute promyelocytic leukemia genome reveals genetic events relevant for disease progression.
Figure 3. Functional validation of the recurrent Jak1 mutation identified in the sequenced mouse APL genome. . From: Sequencing a mouse acute promyelocytic leukemia genome reveals genetic events relevant for disease progression.
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