PMC

Use of window functions. (a) No-windowed MDF . (b) Frequency spectrum of the MDF with no window. (c) MDF using Hann window. (d) DFT of MDF with Hann window. (e) MDF using double-frequency Hann window. (f) DFT of MDF with Hann window.

Scanning electron microscope and spectral analysis of the MDF: (a) Measuring stand; (b) microphotograph of an MDF panel surface; (c) spectra of the outer surface of an MDF panel.

Influence of severity on yield of extract and fiber fraction of (a) poplar; (b) spruce; (c) MDF Sample A; (d) MDF Sample B.

RhoB loss is accompanied by increased γH2AX levels and chromosomal abnormalities. (A) Representative images of WT and RhoB^−/− primary MDF cells after staining for γH2AX. DNA was counterstained with DAPI (blue). Bar, 10 μm. (B) Quantification of the number of γH2AX foci per nucleus. A minimum of 500 nuclei was analyzed per cell type (means ± SD for three independent experiments). (C) Design of the CGH array analysis. Genomic DNA of RhoB^−/− MDF cells was compared to a pool of genomic DNA of MDF cells from four different WT syngeneic mice. As a control, to determine the number of genomic alterations due to interindividual variability, genomic DNA of WT MDF cells was compared to the pool of genomic DNA from WT mice. (D) Table showing whole-genome chromosomal amplification and deletion events as well as the total length (in kbp) of chromosomal alterations in RhoB^−/− MDF cells compared to WT MDF cells analyzed by CGH arrays. The genomic instability index was calculated with the formula (number of deletions + number of amplifications)²/number of altered chromosomes, as previously described (). (E) Chromosome plots for chromosome 15 of RhoB^−/− MDF cells from mouse 2 compared to the pool of WT MDF cells, as determined by a CGH array. This chromosome is representative of the mean numbers of amplification and deletion events in RhoB^−/− MDF cells.