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Results: 5

2.
Figure 4

Figure 4. From: Case-control and prospective studies of dietary ?-linolenic acid intake and prostate cancer risk: a meta-analysis.

Pooled effect of dietary ALA intake on prostate cancer risk in prospective studies. Relative risk with 95% CI, study weights and pooled effect estimates were generated using the general inverse variance method with random effects models (RevMan V.5.1, Cochrane Library software, Oxford, UK). Interstudy heterogeneity was tested by Cochrane's Q (χ2) at a significance level of p<0.10 and quantified by I2, where I2≥50% is considered to be evidence of substantial heterogeneity and ≥75%, considerable heterogeneity.55 ALA, α-linolenic acid.

Amanda J Carleton, et al. BMJ Open. 2013;3(5):e002280.
3.
Figure 2

Figure 2. From: Case-control and prospective studies of dietary ?-linolenic acid intake and prostate cancer risk: a meta-analysis.

Pooled effect of dietary ALA intake on prostate cancer risk in case–control, nested case–control, nested case-cohort and cohort studies. Relative risk with 95% CI, study weights and pooled effect estimates were generated using the general inverse variance method with random effects models (RevMan V.5.1, Cochrane Library software, Oxford, UK). Interstudy heterogeneity was tested by Cochrane's Q (χ2) at a significance level of p<0.10 and quantified by I2, where I2≥50% is considered to be evidence of substantial heterogeneity and ≥75%, considerable heterogeneity.55 ALA, α-linolenic acid.

Amanda J Carleton, et al. BMJ Open. 2013;3(5):e002280.
4.
Figure 3

Figure 3. From: Case-control and prospective studies of dietary ?-linolenic acid intake and prostate cancer risk: a meta-analysis.

Pooled effect of dietary ALA intake on prostate cancer risk in case–control studies. Relative risk with 95% CI, study weights and pooled effect estimates were generated using the general inverse variance method with random effects models (RevMan V.5.1, Cochrane Library software, Oxford, UK). Interstudy heterogeneity was tested by Cochrane's Q (χ2) at a significance level of p<0.10 and quantified by I2, where I2≥50% is considered to be evidence of substantial heterogeneity and ≥75%, considerable heterogeneity.55 ALA, α-linolenic acid.

Amanda J Carleton, et al. BMJ Open. 2013;3(5):e002280.
5.
Figure 5

Figure 5. From: Case-control and prospective studies of dietary ?-linolenic acid intake and prostate cancer risk: a meta-analysis.

Pooled effect of dietary ALA intake on prostate cancer risk in prospective studies after the systematic removal of the study by Giovannucci et al21 following a sensitivity analysis. Relative risk with 95% CI, study weights and pooled effect estimates were generated using the general inverse variance method with random effects models (RevMan V.5.1, Cochrane Library software, Oxford, UK). Interstudy heterogeneity was tested by Cochrane's Q (χ2) at a significance level of p<0.10 and quantified by I2, where I2≥50% is considered to be evidence of substantial heterogeneity and ≥75%, considerable heterogeneity.55 ALA, α-linolenic acid.

Amanda J Carleton, et al. BMJ Open. 2013;3(5):e002280.

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