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Results: 3

1.
Figure 3

Figure 3. From: Reversible electrophysiological abnormalities in acute secondary hyperkalemic paralysis.

The electrocardiogram at admission revealing broad QRS complexes with tall peaked T waves

Karkal R. Naik, et al. Ann Indian Acad Neurol. 2012 Oct-Dec;15(4):339-343.
2.
Figure 2

Figure 2. From: Reversible electrophysiological abnormalities in acute secondary hyperkalemic paralysis.

The F-wave studies in right median nerve at admission (a) and on day 3 after clinical improvement (b) revealing increased latencies of M and F response in the first study which improved after correction of hyperkalemia. Timescale 10ms/d.

Karkal R. Naik, et al. Ann Indian Acad Neurol. 2012 Oct-Dec;15(4):339-343.
3.
Figure 1

Figure 1. From: Reversible electrophysiological abnormalities in acute secondary hyperkalemic paralysis.

Motor nerve conduction studies in right median and ulnar nerves stimulated at wrist (A, D), elbow (B, E) and arm (C, F). At admission, distal latencies are prolonged with reduction of CMAP amplitude and duration on proximal stimulation (1a) which improved after dialysis (1b)

Karkal R. Naik, et al. Ann Indian Acad Neurol. 2012 Oct-Dec;15(4):339-343.

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