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1.
Figure 6

Figure 6. From: Cardiovascular Remodeling in Response to Long-Term Exposure to Fine Particulate Matter Air Pollution.

Total plasma antioxidant capacity in filtered air (FA)-exposed and particulate matter (PM2.5)-exposed mice. Data are expressed as mean±SEM (n=6) and analyzed using a Student t test. P<0.05 was considered statistically significant.

Loren E. Wold, et al. Circ Heart Fail. 2012 July 1;5(4):452-461.
2.
Figure 2

Figure 2. From: Cardiovascular Remodeling in Response to Long-Term Exposure to Fine Particulate Matter Air Pollution.

Histological assessments of filtered air (FA) and particulate matter (PM2.5)-exposed hearts. A, Representative heart tissue sections. B, Quantitative analyses of nucleus/cytoplasm ratio. Data are expressed as mean±SEM (n=5) and analyzed using a Student t test. P<0.05 was considered statistically significant.

Loren E. Wold, et al. Circ Heart Fail. 2012 July 1;5(4):452-461.
3.
Figure 4

Figure 4. From: Cardiovascular Remodeling in Response to Long-Term Exposure to Fine Particulate Matter Air Pollution.

Collagen assessment of filtered air (FA)-exposed and particulate matter (PM2.5)-exposed hearts. A, Representative heart tissue sections stained with Picrosirius red. B, Quantitative assessment of area positive stained for collagen. Data are expressed as mean±SEM (n=5) and analyzed using a Student t test. P<0.05 was considered statistically significant.

Loren E. Wold, et al. Circ Heart Fail. 2012 July 1;5(4):452-461.
4.
Figure 5

Figure 5. From: Cardiovascular Remodeling in Response to Long-Term Exposure to Fine Particulate Matter Air Pollution.

Protein analyses and representative western immune blots from whole heart homogenates of filtered air (FA)-exposed and particulate matter (PM2.5)-exposed mice. A, Collagen I; B, collagen III; C, α-myosin heavy chain (MHC); D, β-MHC; E, ratio α-/β-MHC; and F, SER-CA2a. Data are expressed as mean±SEM (n=5) and analyzed using a Student t test. P<0.05 was considered statistically significant.

Loren E. Wold, et al. Circ Heart Fail. 2012 July 1;5(4):452-461.
5.
Figure 3

Figure 3. From: Cardiovascular Remodeling in Response to Long-Term Exposure to Fine Particulate Matter Air Pollution.

Quantitative cardiac mRNA expression of A, Atrial natriuretic peptide (ANP); B, α-tubulin; C, transforming growth factor (TGF)β; D, osteopontin; E, collagen I, and F, collagen III in filtered air (FA)-exposed and particulate matter (PM2.5)-exposed hearts. Data are expressed as mean±SEM (n=5) and analyzed using a Student t test. P<0.05 was considered statistically significant.

Loren E. Wold, et al. Circ Heart Fail. 2012 July 1;5(4):452-461.
6.
Figure 1

Figure 1. From: Cardiovascular Remodeling in Response to Long-Term Exposure to Fine Particulate Matter Air Pollution.

Isolated myocyte function from mice exposed to filtered air (FA) or particulate matter (PM) for 9 months in the OASIS-1 trailer. A, Representative shortening of FA and PM myocyte contraction. B, Percent peak shortening (%PS) normalized to sarcomere length (SL). C, Time-to-90% peak shortening (TPS90), and D, time-to-90% relengthening (TR90). n=20 to 30 myocytes from 3 to 4 mice per group. Data are expressed as mean±SEM and analyzed using a Student t test. P<0.05 was considered statistically significant.

Loren E. Wold, et al. Circ Heart Fail. 2012 July 1;5(4):452-461.

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