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Results: 5

1.
Figure 2

Figure 2. From: Gut-central nervous system axis is a target for nutritional therapies.

Role of gut-central nervous system axis on inflammatory diseases.

Gustavo D Pimentel, et al. Nutr J. 2012;11:22-22.
2.
Figure 4

Figure 4. From: Gut-central nervous system axis is a target for nutritional therapies.

Summary of main physiological and metabolic alterations that modify the gut sick and health before and after the nutritional therapies.

Gustavo D Pimentel, et al. Nutr J. 2012;11:22-22.
3.
Figure 3

Figure 3. From: Gut-central nervous system axis is a target for nutritional therapies.

Secretion sities of anorexigenic hormones, such as glucagon-like peptide 1 (GLP-1) and peptide YY (PYY) are increased after bariatric surgery.

Gustavo D Pimentel, et al. Nutr J. 2012;11:22-22.
4.
Figure 5

Figure 5. From: Gut-central nervous system axis is a target for nutritional therapies.

Role of intestinal microbiota on development or prevention of inflammatory diseases via dietary fibers, probiotic or prebiotic in the peripheral tissues and central nervous system. ZO-1: Zonula Occludens, JAM-1: juntional adhesion molecule 1.

Gustavo D Pimentel, et al. Nutr J. 2012;11:22-22.
5.
Figure 1

Figure 1. From: Gut-central nervous system axis is a target for nutritional therapies.

Role of gut hormones in fasting (right) and postprandial (left) state. During fasting, occurs greater release of ghrelin from the stomach that acts in arcuate nucleus via vagus nerve stimulating hunger. During the fed state (postprandial), occurs release of anorexigenic hormones (GLP-1, OXM, PYY, CCK and GIP) that reach the arcuate nucleus via the brainstem and vagus nerve, activating the satiety. GLP-1: glucagon-like peptide 1, OXM: oxyntomodulin, PYY: peptide YY, CCK: cholecystokinin, GIP: glucose-dependent insulinotropic polypeptide.

Gustavo D Pimentel, et al. Nutr J. 2012;11:22-22.

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